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<?xml-stylesheet type="text/xsl" media="screen" href="/~d/styles/rss2full.xsl"?><?xml-stylesheet type="text/css" media="screen" href="http://feeds.feedburner.com/~d/styles/itemcontent.css"?><rss xmlns:atom="http://www.w3.org/2005/Atom" xmlns:openSearch="http://a9.com/-/spec/opensearchrss/1.0/" xmlns:georss="http://www.georss.org/georss" xmlns:gd="http://schemas.google.com/g/2005" xmlns:thr="http://purl.org/syndication/thread/1.0" xmlns:feedburner="http://rssnamespace.org/feedburner/ext/1.0" version="2.0"><channel><atom:id>tag:blogger.com,1999:blog-8138663246505221783</atom:id><lastBuildDate>Fri, 23 Mar 2012 06:03:49 +0000</lastBuildDate><category>Male anorexia</category><category>Anorexia teens</category><category>Help with anorexia</category><category>Help for anorexia</category><category>Anorexia facts</category><category>Causes of anorexia nervosa</category><category>Anorexia effects</category><category>Sign of anorexia</category><category>Treatment for anorexia</category><category>Symptoms of anorexia</category><title>AnoV</title><description /><link>http://the-anorexia-nervosa.blogspot.com/</link><managingEditor>noreply@blogger.com (Todr)</managingEditor><generator>Blogger</generator><openSearch:totalResults>95</openSearch:totalResults><openSearch:startIndex>1</openSearch:startIndex><openSearch:itemsPerPage>25</openSearch:itemsPerPage><atom10:link xmlns:atom10="http://www.w3.org/2005/Atom" rel="self" type="application/rss+xml" href="http://feeds.feedburner.com/AnorexiaNervosa" /><feedburner:info uri="anorexianervosa" /><atom10:link xmlns:atom10="http://www.w3.org/2005/Atom" rel="hub" href="http://pubsubhubbub.appspot.com/" /><item><guid isPermaLink="false">tag:blogger.com,1999:blog-8138663246505221783.post-1922342393010733663</guid><pubDate>Thu, 02 Jul 2009 22:30:00 +0000</pubDate><atom:updated>2009-07-02T15:30:02.787-07:00</atom:updated><title>Prevalence of malnutrition in medical and surgical wards of a university hospital</title><atom:summary type="text">Malnutrition is frequently found in hospitals, where is related to poor outcomes. There are contradictory data about if prevalence of malnutrition is greater in surgical or medical patients. The aim of this study is to know the prevalence of malnutrition in both groups of patients.METHODS:The nutritional status of 189 patients from medical and surgical wards was assessed with Subjective Global </atom:summary><link>http://feedproxy.google.com/~r/AnorexiaNervosa/~3/4Vh8MNN7ODw/prevalence-of-malnutrition-in-medical.html</link><author>noreply@blogger.com (Todr)</author><description>&lt;img src="http://feeds.feedburner.com/~r/AnorexiaNervosa/~4/4Vh8MNN7ODw" height="1" width="1"/&gt;</description><feedburner:origLink>http://the-anorexia-nervosa.blogspot.com/2009/07/prevalence-of-malnutrition-in-medical.html</feedburner:origLink></item><item><guid isPermaLink="false">tag:blogger.com,1999:blog-8138663246505221783.post-6664195870262831095</guid><pubDate>Thu, 02 Jul 2009 12:26:00 +0000</pubDate><atom:updated>2009-07-02T05:26:00.852-07:00</atom:updated><title>Self-schemas as predictors of disordered eating behaviors</title><atom:summary type="text">There is broad consensus that the eating disorders of anorexia nervosa and bulimia nervosa stem from fundamental disturbances in identity development, but theoretically based empirical support is lacking.OBJECTIVE:To extend work on the identity impairment model by investigating the relationship between organizational properties of the self-concept and change in disordered eating behaviors (DEB) </atom:summary><link>http://feedproxy.google.com/~r/AnorexiaNervosa/~3/-uvgrGpjqxg/self-schemas-as-predictors-of.html</link><author>noreply@blogger.com (Todr)</author><description>&lt;img src="http://feeds.feedburner.com/~r/AnorexiaNervosa/~4/-uvgrGpjqxg" height="1" width="1"/&gt;</description><feedburner:origLink>http://the-anorexia-nervosa.blogspot.com/2009/07/self-schemas-as-predictors-of.html</feedburner:origLink></item><item><guid isPermaLink="false">tag:blogger.com,1999:blog-8138663246505221783.post-9217775266347114210</guid><pubDate>Thu, 02 Jul 2009 08:20:00 +0000</pubDate><atom:updated>2009-07-02T01:20:00.935-07:00</atom:updated><title>Eating disorders and headache: coincidence or consequence ?</title><atom:summary type="text">The eating disorders anorexia nervosa  and bulimia nervosa are important psychiatric and somatic conditions occurring mainly in young women. The aetiology is unknown, but there are social, biological and psychological factors that play a relevant role in the pathogenesis, along with multiple endocrine abnormalities.Hypothalamic monoamines (especially serotonin), neuropeptides (especially </atom:summary><link>http://feedproxy.google.com/~r/AnorexiaNervosa/~3/-UDKLKryw5U/eating-disorders-and-headache.html</link><author>noreply@blogger.com (Todr)</author><description>&lt;img src="http://feeds.feedburner.com/~r/AnorexiaNervosa/~4/-UDKLKryw5U" height="1" width="1"/&gt;</description><feedburner:origLink>http://the-anorexia-nervosa.blogspot.com/2009/07/eating-disorders-and-headache.html</feedburner:origLink></item><item><guid isPermaLink="false">tag:blogger.com,1999:blog-8138663246505221783.post-4811952646529082238</guid><pubDate>Thu, 02 Jul 2009 03:49:00 +0000</pubDate><atom:updated>2009-07-01T20:49:00.706-07:00</atom:updated><title>Phase I trial of nanoparticle albumin-bound paclitaxel in combination with gemcitabine in patients with thoracic malignancies</title><atom:summary type="text">Nab-paclitaxel has a different toxicity profile than solvent-based paclitaxel including a lower rate of severe neutropenia. This trial was designed to determine the maximum tolerated dose and dose limiting toxicities (DLT) of nab-paclitaxel in combination with gemcitabine.METHODS:Patients were required to have a performance status of 0 to 1, &lt; or = three prior cytotoxic chemotherapy regimens, and</atom:summary><link>http://feedproxy.google.com/~r/AnorexiaNervosa/~3/kma4y8ukGxE/phase-i-trial-of-nanoparticle-albumin.html</link><author>noreply@blogger.com (Todr)</author><description>&lt;img src="http://feeds.feedburner.com/~r/AnorexiaNervosa/~4/kma4y8ukGxE" height="1" width="1"/&gt;</description><feedburner:origLink>http://the-anorexia-nervosa.blogspot.com/2009/07/phase-i-trial-of-nanoparticle-albumin.html</feedburner:origLink></item><item><guid isPermaLink="false">tag:blogger.com,1999:blog-8138663246505221783.post-6485586137813355677</guid><pubDate>Wed, 01 Jul 2009 22:44:00 +0000</pubDate><atom:updated>2009-07-01T15:44:00.581-07:00</atom:updated><title>Illness intrusiveness in anorexia nervosa</title><atom:summary type="text">"Illness intrusiveness" refers to illness-induced lifestyle disruptions. The primary aim of the current study was to compare the level of illness intrusiveness in anorexia nervosa (AN) to that reported in a variety of other chronic medical and psychiatric conditions.A secondary aim was to compare the two subtypes of anorexia nervosa (binge/purge vs. restricting) in terms of the nature and extent </atom:summary><link>http://feedproxy.google.com/~r/AnorexiaNervosa/~3/c4G07_rSCo8/illness-intrusiveness-in-anorexia.html</link><author>noreply@blogger.com (Todr)</author><description>&lt;img src="http://feeds.feedburner.com/~r/AnorexiaNervosa/~4/c4G07_rSCo8" height="1" width="1"/&gt;</description><feedburner:origLink>http://the-anorexia-nervosa.blogspot.com/2009/07/illness-intrusiveness-in-anorexia.html</feedburner:origLink></item><item><guid isPermaLink="false">tag:blogger.com,1999:blog-8138663246505221783.post-5785939472685929642</guid><pubDate>Wed, 01 Jul 2009 08:37:00 +0000</pubDate><atom:updated>2009-07-01T01:37:01.716-07:00</atom:updated><title>Partial laparoscopic splenectomy for splenic abscess</title><atom:summary type="text">Splenic abscess as a presentation of a Salmonella infection is described in children and adults. A combination of antibiotics and splenectomy is the standard treatment. We report a 12-year-old girl admitted to the hospital with fever, abdominal pain, and anorexia.White blood cell count was 17,900/microL and C-reactive protein level was 230 mg/L; abdominal ultrasound and abdominal computed </atom:summary><link>http://feedproxy.google.com/~r/AnorexiaNervosa/~3/qXQ6_OBxw1s/partial-laparoscopic-splenectomy-for.html</link><author>noreply@blogger.com (Todr)</author><description>&lt;img src="http://feeds.feedburner.com/~r/AnorexiaNervosa/~4/qXQ6_OBxw1s" height="1" width="1"/&gt;</description><feedburner:origLink>http://the-anorexia-nervosa.blogspot.com/2009/07/partial-laparoscopic-splenectomy-for.html</feedburner:origLink></item><item><guid isPermaLink="false">tag:blogger.com,1999:blog-8138663246505221783.post-6343197597325085838</guid><pubDate>Tue, 30 Jun 2009 22:33:00 +0000</pubDate><atom:updated>2009-06-30T15:36:41.418-07:00</atom:updated><title>Central versus peripheral antagonism of cannabinoid CB1 receptor in obesity</title><atom:summary type="text">The endogenous cannabinoid system plays an important modulatory role in feeding behaviour and metabolism, acting at both central and peripheral levels.Chronic administration of cannabinoid CB(1) receptor antagonists has been found to be effective in experimental obesity. However, clinically available cannabinoid receptor antagonists are inverse agonists that can target CB(1) receptors located in </atom:summary><link>http://feedproxy.google.com/~r/AnorexiaNervosa/~3/lBQuD1M9kTM/central-versus-peripheral-antagonism-of.html</link><author>noreply@blogger.com (Todr)</author><description>&lt;img src="http://feeds.feedburner.com/~r/AnorexiaNervosa/~4/lBQuD1M9kTM" height="1" width="1"/&gt;</description><feedburner:origLink>http://the-anorexia-nervosa.blogspot.com/2009/06/central-versus-peripheral-antagonism-of.html</feedburner:origLink></item><item><guid isPermaLink="false">tag:blogger.com,1999:blog-8138663246505221783.post-2766671865756542411</guid><pubDate>Tue, 30 Jun 2009 11:33:00 +0000</pubDate><atom:updated>2009-06-30T04:33:01.253-07:00</atom:updated><title>An assessment of daily food intake in participants with anorexia nervosa in the natural environment</title><atom:summary type="text">To examine the caloric intake in women with anorexia nervosa (AN) and how it varies by day as a function of the presence or absence of binge eating and/or purging behaviors.METHOD:Female participants with AN (n = 84, mean age = 24.4, range 18-51) were recruited from three different sites. Data on food intake were obtained through the use of 24-h dietary recall using the Nutritional Data Systems </atom:summary><link>http://feedproxy.google.com/~r/AnorexiaNervosa/~3/NUn1TR8NIz8/assessment-of-daily-food-intake-in.html</link><author>noreply@blogger.com (Todr)</author><description>&lt;img src="http://feeds.feedburner.com/~r/AnorexiaNervosa/~4/NUn1TR8NIz8" height="1" width="1"/&gt;</description><feedburner:origLink>http://the-anorexia-nervosa.blogspot.com/2009/06/assessment-of-daily-food-intake-in.html</feedburner:origLink></item><item><guid isPermaLink="false">tag:blogger.com,1999:blog-8138663246505221783.post-6941133547149492564</guid><pubDate>Tue, 30 Jun 2009 08:35:00 +0000</pubDate><atom:updated>2009-06-30T02:14:07.871-07:00</atom:updated><title>An examination of decision making in bulimia nervosa.</title><atom:summary type="text">Patients with eating disorders favor immediate gratification and ignore long-term negative consequences. This study investigated decision making in bulimia nervosa using the Iowa Gambling Task (IGT) and skin conductance responses (SCR).METHOD:A total of 26 bulimia nervosa patients and 51 healthy controls took part in this study; 29 patients with anorexia nervosa were included for </atom:summary><link>http://feedproxy.google.com/~r/AnorexiaNervosa/~3/4jeAdxlvO3c/examination-of-decision-making-in.html</link><author>noreply@blogger.com (Todr)</author><description>&lt;img src="http://feeds.feedburner.com/~r/AnorexiaNervosa/~4/4jeAdxlvO3c" height="1" width="1"/&gt;</description><feedburner:origLink>http://the-anorexia-nervosa.blogspot.com/2009/06/examination-of-decision-making-in.html</feedburner:origLink></item><item><guid isPermaLink="false">tag:blogger.com,1999:blog-8138663246505221783.post-855995449981485282</guid><pubDate>Fri, 26 Jun 2009 08:13:00 +0000</pubDate><atom:updated>2009-06-26T01:13:02.512-07:00</atom:updated><title>Protein modulates superoxide dismutase and neuropeptide Y-mediated feeding behavior</title><atom:summary type="text">The appetite-suppressing effect of phenylpropanolamine (PPA) has been attributed to its inhibitory action on neuropeptide Y (NPY), an appetite stimulant. However, molecular mechanisms underlying this effect are not clear.This study aimed to investigate if cAMP response element binding protein (CREB) signaling was involved. Moreover, possible role of superoxide dismutase-2 (SOD-2) during PPA </atom:summary><link>http://feedproxy.google.com/~r/AnorexiaNervosa/~3/tN_ai_TQvEU/protein-modulates-superoxide-dismutase.html</link><author>noreply@blogger.com (Todr)</author><description>&lt;img src="http://feeds.feedburner.com/~r/AnorexiaNervosa/~4/tN_ai_TQvEU" height="1" width="1"/&gt;</description><feedburner:origLink>http://the-anorexia-nervosa.blogspot.com/2009/06/protein-modulates-superoxide-dismutase.html</feedburner:origLink></item><item><guid isPermaLink="false">tag:blogger.com,1999:blog-8138663246505221783.post-2431790734012884794</guid><pubDate>Thu, 25 Jun 2009 08:03:00 +0000</pubDate><atom:updated>2009-06-25T01:03:00.812-07:00</atom:updated><title>Temporal occurrence and environmental risk factors</title><atom:summary type="text">Cytauxzoon felis is a tick-transmitted protozoan parasite of domestic and wild felids in the south-central and southeastern United States. Infection of domestic cats (Felis domesticus) with C. felis is typically acute and characterized by fever, anorexia, listlessness, anemia, icterus and usually death within 19-21 days. To determine the temporal occurrence and environmental risk factors </atom:summary><link>http://feedproxy.google.com/~r/AnorexiaNervosa/~3/PS220MvQtUI/temporal-occurrence-and-environmental.html</link><author>noreply@blogger.com (Todr)</author><description>&lt;img src="http://feeds.feedburner.com/~r/AnorexiaNervosa/~4/PS220MvQtUI" height="1" width="1"/&gt;</description><feedburner:origLink>http://the-anorexia-nervosa.blogspot.com/2009/06/temporal-occurrence-and-environmental.html</feedburner:origLink></item><item><guid isPermaLink="false">tag:blogger.com,1999:blog-8138663246505221783.post-5431281865997419051</guid><pubDate>Wed, 24 Jun 2009 08:45:00 +0000</pubDate><atom:updated>2009-06-24T01:45:01.105-07:00</atom:updated><title>Loss of appetite in elderly people</title><atom:summary type="text">The reasons of anorexia of ageing are multiple and not well-known. One of them is loss of appetite. We aimed to know the prevalence of self-reported appetite-loss in the elderly and its relationship with nutritional status, muscle strength and functional capacity.SUBJECTS AND METHOD:A population based cross-sectional study in which 236 non-institutionalized subjects over 70 years were randomly </atom:summary><link>http://feedproxy.google.com/~r/AnorexiaNervosa/~3/qZ4v29U_vMY/loss-of-appetite-in-elderly-people.html</link><author>noreply@blogger.com (Todr)</author><description>&lt;img src="http://feeds.feedburner.com/~r/AnorexiaNervosa/~4/qZ4v29U_vMY" height="1" width="1"/&gt;</description><feedburner:origLink>http://the-anorexia-nervosa.blogspot.com/2009/06/loss-of-appetite-in-elderly-people.html</feedburner:origLink></item><item><guid isPermaLink="false">tag:blogger.com,1999:blog-8138663246505221783.post-5401964577130498168</guid><pubDate>Tue, 23 Jun 2009 08:49:00 +0000</pubDate><atom:updated>2009-06-23T01:49:00.763-07:00</atom:updated><title>Plasma intact fibroblast growth in women with anorexia nervosa</title><atom:summary type="text">Fibroblast growth factor (FGF)23 is a novel phosphaturic factor associated with inorganic phosphate homeostasis. Previous human studies have shown that serum FGF23 levels increase in response to a high phosphate diet.For anorexia nervosa (AN) patients, inorganic phosphate homeostasis is important in the clinical course, such as in refeeding syndrome. The purpose of this study was to determine </atom:summary><link>http://feedproxy.google.com/~r/AnorexiaNervosa/~3/iqjS3t5ErXw/plasma-intact-fibroblast-growth-in.html</link><author>noreply@blogger.com (Todr)</author><description>&lt;img src="http://feeds.feedburner.com/~r/AnorexiaNervosa/~4/iqjS3t5ErXw" height="1" width="1"/&gt;</description><feedburner:origLink>http://the-anorexia-nervosa.blogspot.com/2009/06/plasma-intact-fibroblast-growth-in.html</feedburner:origLink></item><item><guid isPermaLink="false">tag:blogger.com,1999:blog-8138663246505221783.post-3162399893783910643</guid><pubDate>Mon, 22 Jun 2009 14:39:00 +0000</pubDate><atom:updated>2009-06-22T07:44:31.730-07:00</atom:updated><title>MGCD0103 oral dose in patients with advanced solid tumors</title><atom:summary type="text">MGCD0103 is a novel isotype-selective inhibitor of human histone deaceylases (HDACs) with the potential to regulate aberrant gene expression and restore normal growth control in malignancies.PATIENTS AND METHODS:A phase I trial of MGCD0103, given as a three-times-per-week oral dose for 2 of every 3 weeks, was performed in patients with advanced solid tumors. Primary end points were safety, </atom:summary><link>http://feedproxy.google.com/~r/AnorexiaNervosa/~3/EkA65LvU89Y/mgcd0103-oral-dose-in-patients-with.html</link><author>noreply@blogger.com (Todr)</author><description>&lt;img src="http://feeds.feedburner.com/~r/AnorexiaNervosa/~4/EkA65LvU89Y" height="1" width="1"/&gt;</description><feedburner:origLink>http://the-anorexia-nervosa.blogspot.com/2009/06/mgcd0103-oral-dose-in-patients-with.html</feedburner:origLink></item><item><guid isPermaLink="false">tag:blogger.com,1999:blog-8138663246505221783.post-6308780282977645162</guid><pubDate>Mon, 22 Jun 2009 00:56:00 +0000</pubDate><atom:updated>2009-06-21T17:56:00.695-07:00</atom:updated><title>Development and validation of an Eating Disorders Symptom Impact Scale</title><atom:summary type="text">Family members of relatives with eating disorders experience high levels of distress due to the difficulties in their care giving role. However no measures have been developed to measure the specific impact that an individual with an eating disorder has on family life.The aim of this study was to develop a measure to assess the specific caregiving burden of both anorexia nervosa and bulimia </atom:summary><link>http://feedproxy.google.com/~r/AnorexiaNervosa/~3/0OuuKLGBJ1k/development-and-validation-of-eating.html</link><author>noreply@blogger.com (Todr)</author><description>&lt;img src="http://feeds.feedburner.com/~r/AnorexiaNervosa/~4/0OuuKLGBJ1k" height="1" width="1"/&gt;</description><feedburner:origLink>http://the-anorexia-nervosa.blogspot.com/2009/06/development-and-validation-of-eating.html</feedburner:origLink></item><item><guid isPermaLink="false">tag:blogger.com,1999:blog-8138663246505221783.post-3080261338180595688</guid><pubDate>Sun, 21 Jun 2009 14:52:00 +0000</pubDate><atom:updated>2009-06-21T07:52:00.763-07:00</atom:updated><title>Treatment of anorexia nervosa: insights and obstacles</title><atom:summary type="text">Anorexia nervosa is a behavioral disorder characterized by ego-syntonic self-starvation, denial of illness and ambivalence towards treatment. Treatment refusal and drop-out rates are high and relapse is common.Treatment is best viewed as comprised of two phases, weight restoration and normalization of eating behavior followed by relapse prevention. Most patients verbalize a desire to change, </atom:summary><link>http://feedproxy.google.com/~r/AnorexiaNervosa/~3/-EtSb6K7gOE/treatment-of-anorexia-nervosa-insights.html</link><author>noreply@blogger.com (Todr)</author><description>&lt;img src="http://feeds.feedburner.com/~r/AnorexiaNervosa/~4/-EtSb6K7gOE" height="1" width="1"/&gt;</description><feedburner:origLink>http://the-anorexia-nervosa.blogspot.com/2009/06/treatment-of-anorexia-nervosa-insights.html</feedburner:origLink></item><item><guid isPermaLink="false">tag:blogger.com,1999:blog-8138663246505221783.post-1566453952462041484</guid><pubDate>Sun, 21 Jun 2009 11:47:00 +0000</pubDate><atom:updated>2009-06-21T04:47:03.078-07:00</atom:updated><title>Neurobiology of anorexia and bulimia nervosa</title><atom:summary type="text">Anorexia nervosa (AN) and bulimia nervosa (BN) are related disorders of unknown etiology that most commonly begin during adolescence in women. Anorexia nervosa and bulimia nervosa have unique and puzzling symptoms, such as restricted eating or binge-purge behaviors, body image distortions, denial of emaciation, and resistance to treatment.These are often chronic and relapsing disorders, and </atom:summary><link>http://feedproxy.google.com/~r/AnorexiaNervosa/~3/hLj_hQe5_yE/neurobiology-of-anorexia-and-bulimia.html</link><author>noreply@blogger.com (Todr)</author><description>&lt;img src="http://feeds.feedburner.com/~r/AnorexiaNervosa/~4/hLj_hQe5_yE" height="1" width="1"/&gt;</description><feedburner:origLink>http://the-anorexia-nervosa.blogspot.com/2009/06/neurobiology-of-anorexia-and-bulimia.html</feedburner:origLink></item><item><guid isPermaLink="false">tag:blogger.com,1999:blog-8138663246505221783.post-8253284743713241226</guid><pubDate>Sun, 21 Jun 2009 09:20:00 +0000</pubDate><atom:updated>2009-06-21T02:20:01.219-07:00</atom:updated><title>High ambient temperature reverses hypothalamic MC4 receptor overexpression in an animal model of anorexia nervosa</title><atom:summary type="text">The potential involvement of the melanocortin system in the beneficial effects of heat application in rats submitted to activity-based anorexia (ABA), an analogous model of anorexia nervosa (AN), was studied.Once ABA rats had lost 20% of body weight, half of the animals were exposed to a high ambient temperature (HAT) of 32 degrees C, whereas the rest were maintained at 21 degrees C. Control </atom:summary><link>http://feedproxy.google.com/~r/AnorexiaNervosa/~3/-in2WbVUar4/high-ambient-temperature-reverses.html</link><author>noreply@blogger.com (Todr)</author><description>&lt;img src="http://feeds.feedburner.com/~r/AnorexiaNervosa/~4/-in2WbVUar4" height="1" width="1"/&gt;</description><feedburner:origLink>http://the-anorexia-nervosa.blogspot.com/2009/06/high-ambient-temperature-reverses.html</feedburner:origLink></item><item><guid isPermaLink="false">tag:blogger.com,1999:blog-8138663246505221783.post-5511742263841499032</guid><pubDate>Sat, 20 Jun 2009 18:18:00 +0000</pubDate><atom:updated>2009-06-20T11:18:00.854-07:00</atom:updated><title>Tonic and phasic effects of corticosterone on food restriction</title><atom:summary type="text">In the experimental rat model of anorexia nervosa the interactions between the hyperactivity of the hypothalamo-pituitary-adrenal (HPA) axis and increased physical activity associated with food restriction remain unidentified.In addition to their role in energy homeostasis, glucocorticoids have complex effects in the central nervous system, increasing the salience of activities such as wheel </atom:summary><link>http://feedproxy.google.com/~r/AnorexiaNervosa/~3/t1Xtomxv3-A/tonic-and-phasic-effects-of.html</link><author>noreply@blogger.com (Todr)</author><description>&lt;img src="http://feeds.feedburner.com/~r/AnorexiaNervosa/~4/t1Xtomxv3-A" height="1" width="1"/&gt;</description><feedburner:origLink>http://the-anorexia-nervosa.blogspot.com/2009/06/tonic-and-phasic-effects-of.html</feedburner:origLink></item><item><guid isPermaLink="false">tag:blogger.com,1999:blog-8138663246505221783.post-2931722066467987570</guid><pubDate>Sat, 20 Jun 2009 16:13:00 +0000</pubDate><atom:updated>2009-06-20T09:13:00.738-07:00</atom:updated><title>Mechanisms of cancer cachexia</title><atom:summary type="text">Up to 50% of cancer patients suffer from a progressive atrophy of adipose tissue and skeletal muscle, called cachexia, resulting in weight loss, a reduced quality of life, and a shortened survival time. Anorexia often accompanies cachexia, but appears not to be responsible for the tissue loss, particularly lean body mass. An increased resting energy expenditure is seen, possibly arising from an </atom:summary><link>http://feedproxy.google.com/~r/AnorexiaNervosa/~3/CD7le77_4hc/mechanisms-of-cancer-cachexia.html</link><author>noreply@blogger.com (Todr)</author><description>&lt;img src="http://feeds.feedburner.com/~r/AnorexiaNervosa/~4/CD7le77_4hc" height="1" width="1"/&gt;</description><feedburner:origLink>http://the-anorexia-nervosa.blogspot.com/2009/06/mechanisms-of-cancer-cachexia.html</feedburner:origLink></item><item><guid isPermaLink="false">tag:blogger.com,1999:blog-8138663246505221783.post-5417327454758059200</guid><pubDate>Sat, 20 Jun 2009 11:06:00 +0000</pubDate><atom:updated>2009-06-20T04:06:01.568-07:00</atom:updated><title>Systematic review and meta-analysis of the baseline concentrations and physiologic responses of gut hormones to food in eating disorders</title><atom:summary type="text">Disturbances in gastrointestinal hormones have been widely identified in persons with eating disorders (EDs) and have been implicated in their clinical pathologies.OBJECTIVE:The objective was to identify, critically examine, and summarize studies investigating the short-term response of gastrointestinal hormones to food in persons with an ED, including the subtypes anorexia nervosa and bulimia </atom:summary><link>http://feedproxy.google.com/~r/AnorexiaNervosa/~3/rUCkDjYFVto/systematic-review-and-meta-analysis-of_20.html</link><author>noreply@blogger.com (Todr)</author><description>&lt;img src="http://feeds.feedburner.com/~r/AnorexiaNervosa/~4/rUCkDjYFVto" height="1" width="1"/&gt;</description><feedburner:origLink>http://the-anorexia-nervosa.blogspot.com/2009/06/systematic-review-and-meta-analysis-of_20.html</feedburner:origLink></item><item><guid isPermaLink="false">tag:blogger.com,1999:blog-8138663246505221783.post-2310554256115166094</guid><pubDate>Sat, 20 Jun 2009 09:12:00 +0000</pubDate><atom:updated>2009-06-20T02:12:01.662-07:00</atom:updated><title>Leptin in humans: lessons from translational research</title><atom:summary type="text">Leptin has emerged over the past decade as a key hormone in not only the regulation of food intake and energy expenditure but also in the regulation of neuroendocrine and immune function as well as the modulation of glucose and fat metabolism as shown by numerous observational and interventional studies in humans with (complete) congenital or relative leptin deficiency.These results have led to </atom:summary><link>http://feedproxy.google.com/~r/AnorexiaNervosa/~3/tEZbwKrqVgw/leptin-in-humans-lessons-from.html</link><author>noreply@blogger.com (Todr)</author><description>&lt;img src="http://feeds.feedburner.com/~r/AnorexiaNervosa/~4/tEZbwKrqVgw" height="1" width="1"/&gt;</description><feedburner:origLink>http://the-anorexia-nervosa.blogspot.com/2009/06/leptin-in-humans-lessons-from.html</feedburner:origLink></item><item><guid isPermaLink="false">tag:blogger.com,1999:blog-8138663246505221783.post-4139993669956219887</guid><pubDate>Fri, 19 Jun 2009 21:21:00 +0000</pubDate><atom:updated>2009-06-19T14:21:00.327-07:00</atom:updated><title>AdipoR1 mediates the anorexigenic and insulin/leptin-like actions of adiponectin in the hypothalamus</title><atom:summary type="text">Adiponectin exerts an insulin-sensitizing effect, improving insulin action in peripheral tissues and restraining insulin resistance. Here, we explore the hypothesis that adiponectin can reproduce some of the actions of insulin/leptin in the hypothalamus.The presence of AdipoR1 and AdipoR2 was mapped to the arcuate and lateral hypothalamic nuclei. Icv adiponectin reduced food intake, which was </atom:summary><link>http://feedproxy.google.com/~r/AnorexiaNervosa/~3/YsM4pH4x8J4/adipor1-mediates-anorexigenic-and.html</link><author>noreply@blogger.com (Todr)</author><description>&lt;img src="http://feeds.feedburner.com/~r/AnorexiaNervosa/~4/YsM4pH4x8J4" height="1" width="1"/&gt;</description><feedburner:origLink>http://the-anorexia-nervosa.blogspot.com/2009/06/adipor1-mediates-anorexigenic-and.html</feedburner:origLink></item><item><guid isPermaLink="false">tag:blogger.com,1999:blog-8138663246505221783.post-3917215952845786841</guid><pubDate>Fri, 19 Jun 2009 18:17:00 +0000</pubDate><atom:updated>2009-06-19T11:17:00.310-07:00</atom:updated><title>Clinical features of 107 autoimmune hepatitis patients and 30 of them with AIH-primary biliary cirrhosis overlap syndrome</title><atom:summary type="text">In order to provide a reliable basis for the diagnosis and treatment of autoimmune hepatitis (AIH) and its overlap syndrome, we investigated the clinical, immunological characteristics of and the therapeutic methods for AIH and AIH-primary biliary cirrhosis (PBC) overlap syndrome.METHODS:One hundred seven patients (77 with AIH and 30 with AIH-PBC overlap syndrome) were enrolled in the study. </atom:summary><link>http://feedproxy.google.com/~r/AnorexiaNervosa/~3/cpfvBv9xZlU/clinical-features-of-107-autoimmune.html</link><author>noreply@blogger.com (Todr)</author><description>&lt;img src="http://feeds.feedburner.com/~r/AnorexiaNervosa/~4/cpfvBv9xZlU" height="1" width="1"/&gt;</description><feedburner:origLink>http://the-anorexia-nervosa.blogspot.com/2009/06/clinical-features-of-107-autoimmune.html</feedburner:origLink></item><item><guid isPermaLink="false">tag:blogger.com,1999:blog-8138663246505221783.post-7145501337426169015</guid><pubDate>Fri, 19 Jun 2009 12:13:00 +0000</pubDate><atom:updated>2009-06-19T05:13:01.927-07:00</atom:updated><title>Mental disorders among relatives of patients with anorexia nervosa and bulimia nervosa</title><atom:summary type="text">Family studies of anorexia (AN) and bulimia (BN) nervosa in relatives of patients with eating disorders compared to control subjects are rare in German-speaking countries.METHODS:A German multicenter study compared first-, second- and third-degree relatives of 65 adolescent anorexia  nervosa subjects (n = 746), 21 adolescent  bulimia nervosa subjects (n = 265) and relatives of 11 adolescent </atom:summary><link>http://feedproxy.google.com/~r/AnorexiaNervosa/~3/tf7BDQlkQ40/mental-disorders-among-relatives-of.html</link><author>noreply@blogger.com (Todr)</author><description>&lt;img src="http://feeds.feedburner.com/~r/AnorexiaNervosa/~4/tf7BDQlkQ40" height="1" width="1"/&gt;</description><feedburner:origLink>http://the-anorexia-nervosa.blogspot.com/2009/06/mental-disorders-among-relatives-of.html</feedburner:origLink></item></channel></rss>

