droopyyoupi

Blogiversaire jour 1

2012-01-31T18:38:00.000+01:00

http://www.littlemissbeauty.fr/wordpress/2012/01/edenens-huile-demaquillante-lactee-nectar-originel/

un médecin traitant lyme se voit "mis à pied "

2012-01-31T14:57:00.000+01:00

Wisconsin Lyme Doctor Gets Reprieve
Submitted by Fran Zell on Sun, 01/29/2012 - 9:10pm

It has the makings of a John LeCarré thriller: A debilitating, potentially deadly, infectious disease carried by an insect the size of a poppy seed is spreading across the land. The medical establishment insists that everything is under control and that all these “sick” people are slackers or need psychiatric help. A few doctors—just a few—believe the people are really sick, and treat them according to guidelines not approved by insurance companies. One such doctor gets people in wheelchairs walking again, gives bedridden folks a new lease on life. Sick people flock to him from all over the land. He has saved many lives, and never harmed a patient. Yet one day his license is summarily suspended. There are not enough other doctors in the land to treat his many patients.
This is a story about that doctor and a state Medical Examining Board that has not always looked kindly on practitioners who treat “outside the box.” The disease is real. It’s called Lyme, and by some expert accounts, it’s an epidemic. The place is Wisconsin. But it could happen almost anywhere.

[Photos: Dr. Robert Waters in his Wisconsin Dells office; Lyme Patient John Blohm; Atty. Arthur Thexton at the November hearing for Dr. Hoffmann.]

Wisconsin Lyme doctor gets reprieve
By Fran Zell and Tom Boswell
John Blohm was a charter airplane pilot until a mysterious disease made it impossible for him to fly. Neurologists did meticulous work-ups and found nothing wrong, despite the fact that Blohm couldn’t hear or see right, suffered from exhaustion, extreme dizziness, bouts of fever and chills, tremors, and what he refers to as “lightning-bolt” zaps of pain.
“I spiraled downhill for years,” says Blohm. “The doctors referred me to psychiatrists and I began to think I was crazy. A Mayo Clinic specialist accused me of malingering and said I should go back to work.”
After seven years, Blohm did some research and realized his symptoms were typical of Lyme disease, a tick-borne bacterial infection endemic to Wisconsin. He remembered that years earlier – just before his symptoms began – he had been bitten by a tick in his suburban Beloit backyard. He had tested negative for Lyme during the course of his illness but now he found a doctor who treated chronic Lyme disease at his small clinic in Waupaca.
“Dr. Hoffmann was a godsend,” he says. “By the time I first saw him, I’d lost the use of one arm and could barely walk. The vertigo was unbearable. I was losing about five pounds a week. He diagnosed me with Lyme based on my symptoms and history and started treating me with antibiotics. He brought me back from the edge.”
In mid-November, Blohm’s treatment ended abruptly. His Lyme doctor — John Gregory Hoffmann — was summoned to Madison by the State’s Medical Examining Board. After a brief hearing, his medical license was suspended, effective immediately.
“The state says this has nothing to do with Lyme disease, but I think it’s a veiled attack,” asserted Blohm, who spearheaded a movement among fellow Lyme sufferers to challenge the Board’s decision. “Dr. Hoffmann does not treat Lyme according to (standard) guidelines, and I think they’re trying to stop him.”
Arthur Thexton, the Division of Enforcement (DOE) prosecuting attorney who filed the complaint requesting summary suspension, would neither confirm nor deny Blohm’s suspicion. “If I want to keep my job, I can’t talk to the press,” he said.
On January 18 Hoffmann, his attorney, and the Medical Board reached an agreement whereby the suspension of Hoffmann’s license would be “stayed” subject to various terms and conditions. Hoffmann must work under supervision of a professional mentor, may not accept new patients, must complete a “robust self-study program,” and pass a Special Purpose Examination (SPEX), a test used by medical boards to re-examine a licensed or previously licensed physician’s basic medical knowledge.
“They’re saying I’m not up to standard with modern medical practice, that I practice out of the routine.” Hoffmann commented after his January Board appearance. “It’s not a complaint regarding patient care.”
Thexton reopened an old (1997) case on Hoffmann in 2007, for reasons that are not revealed in the case file. The old case related to a 1992 incident in which Hoffmann had been drinking while on call. The case was fully resolved in 2006.
Marina Andrews, who blogs about Lyme from Luck, Wisconsin, said the new case originated with a former patient who complained that Hoffman would not prescribe pain medication during an office visit. Thexton then asked for patient records, many of them Lyme files. “There was a public outcry at the time and Thexton was temporarily removed from the case,” Andrews said.
In 2008, the Board ruled that Hoffmann’s charts suggested possible “deficiencies in medical knowledge and practice standards” and referred him to the UW Medical Physician Assessment Center. Last August, a report from UW said Hoffmann had “significant deficiencies” based on standardized testing and recommended more testing. Thexton called for suspension, contending there was an immediate need to protect the public from Hoffmann.
Maybe he didn’t study for the test, but he’s a fabulous doctor, said Hoffmann patient Jennifer Christie of De Pere. “I had a severe situation recently. My kidneys were bleeding. I was in the ER three days in a row. Everyone at Bellin (Medical Center in Green Bay) missed it. I made one call to Doc and he immediately knew it was a drug interaction related to a med my primary provider put me on.”
“Dr. Hoffmann saved my life,” said Lyme patient Della Haugen, a former photojournalist for a Madison TV station, and one of two thousand people who wrote letters and signed a petition of support prior to the November hearing. “I saw lots of UW doctors and none of them could help me. I got so sick I had to stop working.”
A Milwaukee area public official who testified at the November hearing said Hoffmann’s Lyme treatment enabled her to leave her wheelchair. She told a reporter that since being bitten by a tick 14 years ago, she endured fainting spells, horrible neck and eye pain and numbness on one side of her body. “Then my legs wouldn’t support me. I was writing my will,” she said of the time before she started seeing Hoffmann.
Dr. Gene Musser, the Board member who made the motion to suspend Hoffmann’s license, said he could not discuss the case other than to note that the Board did not sanction Hoffmann because of his Lyme treatment practices.
But the fact remains that Lyme is a major medical controversy, to the point that Hoffmann was treating a disease — chronic Lyme — that the academic-medical establishment has long insisted does not exist.
“Is this an issue the state has with him because he’s operating outside of medical thought? I think it is,” said Dr. Robert Waters, who operates a medical clinic in the Dells. “They’re holding him responsible for something that happened 20 years ago. This doesn’t have anything to do with the sick people he treats. There’s no evidence he harmed anyone.
“The state has unlimited funds to continue a prosecution,” Waters said. “When a doctor runs out of money, his lawyers ask him to settle and stipulate and admit to certain things (he’s) done ‘wrong.’ The burden of proof is on the doctor, even when there are no complaints from patients.”
Waters said that the state, and Thexton in particular, have a pattern of coming down hard on alternative health care practitioners. He himself was the center of a highly publicized investigation by Thexton that was dismissed as groundless, in 2003, shortly after Thexton was removed from the case. “It went on for six and a half years, cost me $50,000 in legal fees, untold stress, heartache and lost income,” he said.
Thexton and DOE attorney James Polewski investigated so many alternative practitioners at the time that some of those involved, including Watertown nurse Barbara Lemke, considered it a “vendetta.” Lemke was called before the Board for using an allergy elimination treatment called acupressure. She ultimately appealed her case and was exonerated. “But it cost me $26,000 (in attorney fees) to keep my license,” she said.
As a result of the experience, Lemke helped found the Wisconsin Association for Health Freedom. “We're currently trying to get a bill passed that would allow practitioners to use alternative therapies without fear of being prosecuted,” she said.
Waters was investigated for using a toxin-removing therapy called chelation for a controversial purpose — treating cardiovascular disease and circulatory disorders. Thexton publicly called it “quackery,” even though the National Institute for Health was conducting a major study on chelation in which Waters had been asked to participate.
Hoffmann follows treatment protocols for Lyme developed by the International Lyme and Associated Disease Society (ILADS), a small, but vocal medical group based in Washington, D.C. ILADS contends that, if untreated or under-treated, Lyme bacteria remain in the body and the disease goes “chronic,” manifesting in a host of serious symptoms often misdiagnosed as fibromyalgia, chronic fatigue, rheumatoid arthritis, early Alzheimer’s, Parkinson’s, multiple sclerosis, Crohn’s disease, ALS and more.
Most doctors – and health insurers – debunk ILADS. They follow Lyme protocols mandated by the Infectious Disease Society of America (IDSA), which holds that Lyme bacteria are eradicated after a short course of antibiotics.
“If symptoms remain after that, they say it’s a somatic syndrome,” said Dr. Burton Waisbren Sr., a founding member of IDSA who disagrees with the group’s position on Lyme. The Milwaukee internist and infectious disease specialist asserts that chronic Lyme does exist, and that patients seriously ill with it have responded to long-term antibiotic therapy at his clinic. In his new book, Treatment of Chronic Lyme Disease (iUniverse, Inc.), Waisbren chronicles 51 recent cases (not all of them successful) that have led him to conclude that chronic Lyme is a national epidemic.
Recent statistics from the Centers for Disease Control (CDC) indicate that Wisconsin is fast becoming ground zero for Lyme disease. Reported incidences have increased more than five-fold since 2000, making Wisconsin the third hardest-hit state in the country. But there are only a handful of doctors in the Midwest who will treat Lyme outside of IDSA guidelines.
“I think for anyone in Wisconsin prescribing outside of standard protocols, there is always going to be the chance of an issue coming up,” said Kim Saxe, a naturopathic doctor who provides complementary services to many Lyme patients at her Brookfield wellness center.
Saxe said that insurance companies “are raising red flags” and reporting doctors for their prescribing methods. “It’s been an issue with Lyme doctors in other parts of the country who have lost their licenses,” she said. “It’s how the issue reaches medical boards. It is also why many Lyme doctors are choosing not to bill insurance.
“I know there were other claims made against Dr. Hoffmann,” she said, “but I find it a little challenging to believe (the case) was unrelated to his Lyme prescribing.”
Saxe said she knew little about Lyme until she contracted it herself about four years ago. She said that for two years she saw a lot of doctors and spent $20,000 out of pocket on tests that turned up nothing, despite her abdominal dysfunction and loss of muscle mass.
“No one thought of Lyme. Doctors told me I needed antidepressants and anxiety medication, but I knew something serious was going on.”
Eventually she ran her own Lyme test. “Lyme testing is horrible, she noted. “There are many false negatives.” But Saxe tested positive, then found a Milwaukee internist who treats Lyme outside of IDSA guidelines but does not publicize the fact for fear of losing his license.
“Lyme doctors are doing what they feel and know is right for patients, even though it is not the standard of care,” she said.
Waters went through ILADS training in 2009 and now also treats Lyme. He said he stopped taking Medicare, Medicaid, and private insurance years ago because he didn’t want to risk being investigated by the state for doing “what I know is right for patients.” The complaint Thexton prosecuted him for originated with a doctor and Blue Cross Blue Shield.
State Senator Dale Schultz (R) supported Waters at the time and said he still does.
“I have a number of constituents who are his chelation patients and they are passionate about him,” he said. “That led me to be supportive too. Information came to light that the U.S. government was using chelation to take bad things out of the body. It seemed extremely unreasonable to me for the state to pick on someone for doing the same treatment.”
Blue Cross Blue Shield also initiated the complaint that precipitated a nine-year licensing battle that Dr. Eleazer Kadile, a Green Bay chelation practitioner, said cost him $500,000 in legal bills.
“It was purely politics,” alleges Kadile, who was ultimately cleared on charges relating to chelation. Thexton’s key witness in both chelation cases was a Massachusetts doctor with consulting ties to the insurance industry. He was largely discredited on the stand, though he billed the state for nearly $86,000.
Sheldon Wasserman, a Milwaukee physician on the Medical Examining Board, believes that some of the conflict over state regulation of alternative health care practitioners can be mitigated with a re-write of Chapter Med 10 of the Administrative Code, which covers unprofessional conduct of physicians. Wasserman, who served 14 years in the State Assembly (D), assumed the chair position on the Board in January.
“Rewriting of Chapter 10 will be one of my top priorities for the year,” Wasserman said. He feels there is an “emerging position” on the Board to develop rules that would allow physicians freedom to practice as long as there was “truly-given informed consent” on the part of patients. The code revision would “put a lot of burden on physicians” to inform patients that their treatment was not “the standard of care” and “basically create a buyer-beware type of situation.
“I think people have the right to decide what they want, and the right to do what they want with their own bodies, across the board,” commented Wasserman.
Waters addressed the issue of doctor-patient understanding several years ago, following the lead of alternative practitioners in other states. He turned his clinic into a private association, charging a nominal fee to patients who then sign a contract accepting services. It enables him to offer the treatments patients seek without interference from governmental bodies, he explained.
John Blohm, for one, said he knew what he was getting into when he started seeing Hoffmann. “He told me about the risks of long-term antibiotic treatment. But I also know it will kill me if I don’t do it. I made a very informed decision. I chose to follow ILADS guidelines.”
The fallout from Hoffmann’s license suspension has been huge, said Kristin Collins, a Pewaukee nurse whose two small children are Hoffmann’s patients.
“He has about 1,500 patients, said Collins, who volunteered at the Waupaca clinic for four days following the Board’s November decision to suspend Hoffmann’s license. “Dr. Hoffmann has his own unique way of treating. He took Medicare and Medicaid, and most other insurance. He never turned his back on anyone because of money. There aren’t enough other Lyme doctors in the state to take on these patients.
“Some people called the office, sobbing,” she said. “They had seen numerous doctors who ignored them. Doc believed them and helped them.”
The “interim” stipulation agreed to by Hoffmann and the Medical Board on January 18 represents a “pathway” to full re-instatement of the doctor’s license, according to Greg Gasper, spokesperson for Wisconsin’s new Department of Safety and Professional Services.
About a half-dozen of Hoffmann’s patients spoke at the meeting in support of him, despite the fact that his case was not listed on the public agenda.
“They (the Board) got the message,” Hoffmann commented.
# # #
Photos of Atty. Arthur Thexton and Dr. Robert Waters by Tom Boswell. Photo of John Blohm by Laura Blohm.
About the writers: Fran Zell and Tom Boswell are freelance journalists residing in Evansville, Wisconsin. Zell has written extensively about Lyme disease, including stories in the Chicago Tribune and Milwaukee Magazine. She has written cover stories for Isthmus and the Chicago Reader. Tom Boswell is a writer and photographer who has had cover stories in the National Catholic Reporter, Shepherd Express and other publications. His photographs for an Isthmus cover story in December 1996, won a first place award from the Milwaukee Press Club.

les mycoplasmes dans la maladie de lyme chronique

2012-01-31T11:20:00.000+01:00

Mycoplasma - Often Overlooked In Chronic Lyme Disease
by Scott Forsgren

Those of us with chronic Lyme disease are quite familiar with the names of the better known Lyme co-infections. Babesia, Bartonella, and Ehrlichia have become everyday words. As much as we would like to rid ourselves of these illness-producing pathogens, they have become a part of our daily struggle to regain a sense of health and wellness. Unfortunately, these are not the only co-infections seen in chronic Lyme disease. For some reason, Mycoplasma infections are not only lesser known by patients, but seemingly often overlooked by doctors as well. It is important for us, as patients, to educate ourselves on the topic of Mycoplasma and to ask our practitioners how we are being evaluated and treated for these infections.
In 1987, Dr. Garth Nicolson, PhD was a professor at the University of Texas at Houston when his wife, an instructor at Baylor College of Medicine, became seriously ill and nearly died. She was diagnosed with a Mycoplasma infection, treated, and later recovered. A few years later, their daughter, who had served in the Gulf War, returned from active duty quite ill. Not only was she sick, but the symptoms that she exhibited were very similar to those that Dr. Nicolson's wife had expressed years earlier.

At that point, Dr. Nicolson had the idea that his daughter's illness could be the result of an infection and started to investigate his theory further. As his work progressed, he looked at Brucella, Borrelia, Ehrlichia, and other chronic intracellular infections that have the potential to cause illness and present with overlapping signs and symptoms. In Gulf War veterans that were being evaluated, approximately 45% of those that were ill had Mycoplasma infection. It was found that the infection was a particular type of Mycoplasma, namely a peculiar species called Mycoplasma fermentans.
Very little was known about this particular species of Mycoplasma at the time except that the Armed Forces Institute of Pathology and the Army had been doing research on the organism. Once this likely causative agent of Gulf War Illness (GWI) had been identified in about one-half of the GWI cases, Dr. Nicolson recommended that the Mycoplasma-infected Gulf War veterans be treated with Doxycycline. He then found himself the target of vicious attacks for making the connection between the illness and M. fermentans. Dr. Nicolson shared that "even talking about this organism was highly discouraged." In fact, until the Gulf War, the military's own medical school had been teaching about the dangers of M. fermentans for years.

Background

Just years earlier in Texas, prisons emerged in which many of the inmates and guards came down with neurodegenerative conditions at rates that were far from ordinary. In Huntsville, where three large State prisons are found, there were about 70 cases of ALS, numerous cases of Multiple Sclerosis, and highly unexpected numbers of Rheumatoid Arthritis cases. At that time, the term "Mystery Disease" was used to identify the unusual illnesses that so many seemed to have acquired.
Dr. Nicolson started testing prison guards and their family members and found that very high numbers of these people were testing positive for Mycoplasma fermentans. Furthermore, this appeared to be a weaponized version of the organism called M. fermentans incognitus, a specific strain of Mycoplasma that had been altered to cause more severe symptoms, to be more virulent, and to be more survivable than the naturally occurring M. fermentans. Dr. Nicolson believed that biological weapons experiments had been carried out on inmates in the Texas prison system for years in which humans had been used as guinea pigs.
As time progressed, these illnesses did not remain confined to the prisoners. Soon after the prisoners unknowingly became a part in these experiments, the prison guards became ill. Their illnesses gradually became those of their families. It was not long before these Mycoplasma-based illnesses became a broader part of the surrounding Huntsville, Texas landscape.

The Texas prisoners that came down with Amyotrophic Lateral Sclerosis (ALS) later died. In the state of Texas, at the time, the state law dictated that all prisoners that died were later to be autopsied at University of Texas at Galveston. However, that was not what was happening to the prisoners who had died as a result of this horrific experimentation, according to Dr. Nicolson. Through one of his former students who at the time was responsible for the autopsy service at UT Galveston, Dr. Nicolson learned that none of the bodies had been sent there. Dr. Nicolson had discovered that at least six private autopsies a week were being performed on deceased prisoners at a US Army base. The bodies were then sent to a private crematory at a secret location in central Texas. Additionally, prisoner records were destroyed. All of this, according to Dr. Nicolson, violated state law.

Though much of the evidence of this experimentation had been destroyed, a document was found in the basement of an Austin building that was viewed as the "smoking gun". The document indicated that the Texas Prison Board, Baylor College of Medicine, and the Department of Defense were all a part of the experiments involving the Texas prisoners - experiments that later resulted in the death of many of the inmates. According to Dr. Nicolson, some of the experiments used Mycoplasma while others utilized various "cocktails of microbial agents" such as Mycoplasma, Brucella, and DNA viruses such as Parvovirus B19. This project later became the topic of a book by Dr. Nicolson entitled Project Day Lily.

Dr. Nicolson believes that Mycoplasma fermentans is a naturally occurring microbe. However, some of the strains that exist today have been weaponized. Dr. Nicolson's research found unusual genes in M. fermentans incognitus that were consistent with a weaponized form of the organism. Weaponzing of an organism is done in an attempt to make a germ more pathogenic, immunosuppressive, resistant to heat and dryness, and to increase its survival rate such that the germ could be used in various types of weapons. Genes which were part of the HIV-1 envelope gene were found in these Mycoplasma. This means that the infection may not give someone HIV, but that it may result in some of the debilitating symptoms of the HIV disease. Indicators of a weaponized organism were evident in the prison guards in Huntsville as well as in military personnel that were likely exposed to the infections both through military vaccinations as well as through weapons used in the Gulf War.

The unfortunate reality according to Dr. Nicolson is that "once these things get out, you can't put the genie back in the bottle". Once these germs have been released, they are airborne infections that slowly penetrate into the population. In the case of Mycoplasma fermentans, Dr. Nicolson believes that this is exactly what happened. It may be this weaponized form of Mycoplasma that has led to the significant increases in neurodegenerative and autoimmune diseases over the last several years. Those patients with weaponized strains of these organisms are generally very sick. They may experience 60-75 signs and symptoms and are even at risk of their diseases becoming fatal.

In looking at the source of infection in the Gulf War veterans who were contracting Mycoplasma, Dr. Nicolson suggests that vaccinations appear to be the most likely mechanism through which the veterans became infected. Many military personnel that later became ill were far from the battlefields or had received the vaccinations and were never deployed. However, biological weapons sprayers were known to have been deployed by the Iraqis in the Gulf War and were used to spray the sand in Iraq and Kuwait. Gerald Schumacher, a Special Forces colonel in charge of biological weapons detection, blew the whistle on this after he retired. During the Gulf War, his group was not allowed to deploy their biological weapons detectors which led to reports that no such weapons were detected or used.
The Iraqis received a great deal of assistance on biological warfare from the United States during the Iran-Iraq Conflict. Both chemical and biologic weapons were given to them from the United States. After the Gulf War, rather than taking inventory of these weapons, they were blown up. Dr. Nicolson indicates that some of his patients have taken videos standing next to crates with Hazardous Materials tags from the United States. In the same videos, the crates are opened and weapons are clearly striped as having originated from the United States and being both chemical and biological weapons.

There were clear indicators that Iraq had offensive weapons in their arsenal. In Kuwait, many people had become quite ill. It was estimated that 25% of the population after the Gulf War had signs and symptoms which matched the symptoms of those infected with weaponized Mycoplasma. There were also a number of other chemical exposures and thus, there was never a clear indicator as to whether or not the Iraqi illnesses were caused by biologic or chemical agents.

When asking Dr. Nicolson how much he personally has been harassed for bringing much of this information to light, he shared that it has been "a horrific time". After Dr. Nicolson exposed the Huntsville prison experiments, the University of Texas educational system attempted to fire him from his tenured and highly respected position. Dr. Nicolson shared that a tremendous amount of pressure was put on the University of Texas system to "shut him up and close his laboratory". He was threatened on an almost daily basis with closing his lab as he continued to do his research on Mycoplasma. This became a major subject in the book Project Day Lily. Fortunately, for many of us struggling with chronic illnesses, Dr. Nicolson's experience and knowledge continue to be a benefit in that we understand so much more than we otherwise would about this formidable foe called Mycoplasma.

Symptoms

The signs and symptoms of Mycoplasma infection are highly variable and thus it is not uncommon for a diagnosis to be entirely missed. A partial list of symptoms includes chronic fatigue, joint pain, intermittent fevers, headaches, coughing, nausea, gastrointestinal problems, diarrhea, visual disturbances, memory loss, sleep disturbances, skin rashes, joint stiffness, depression, irritability, congestion, night sweats, loss of concentration, muscle spasms, nervousness, anxiety, chest pain, breathing irregularities, balance problems, light sensitivity, hair loss, problems with urination, congestive heart failure, blood pressure abnormalities, lymph node pain, chemical sensitivities, persistent coughing, eye pain, floaters in the eyes, and many others. On Dr. Nicolson's web site at http://www.immed.org, a full list of signs and symptoms and an illness survey form can be found.

It doesn't take long to see that the symptoms of Mycoplasma infections are very similar to the symptoms of Borrelia infections in chronic Lyme disease. Dr. Nicolson has looked at some of the more common neurodegenerative diseases and the infections that are associated with each. Mycoplasma is commonly found in patients with ALS, Multiple Sclerosis, Autism, Chronic Fatigue Syndrome, Rheumatoid Arthritis, Chronic Asthma, Lyme disease, and many other chronic disease conditions.

Characteristics

Mycoplasma are pleomorphic bacteria which lack a cell wall and, as a result, many antibiotics are not effective against this type of bacteria. There are over 100 known species of Mycoplasma, but only a half dozen or so are known to be pathogenic in humans. The pathogenic species are intracellular and must enter cells to survive. Once they are inside the cells, they are not recognized by the immune system and it is difficult to mount an effective response.

They stimulate reactive-oxygen species (ROS) which damage cell membranes. They release toxins into the body. Infected cells can be stimulated to undergo programmed cell death which may result in ALS or other severe neurological presentations. 90% of ALS patients evaluated were found to have Mycoplasma infections, whereas Mycoplasma was found in 100% of ALS patients with Gulf War Syndrome, almost all of which were weaponized M. fermentans incognitus.

They are thought of as "borderline anaerobes", meaning that they generally prefer low oxygen environments. Dr. Nicolson has found that airline employees are much more susceptible to these types of infections and that symptoms worsen with frequent long flights at low oxygen tension. Mycoplasma also have some characteristics of viruses.

Mycoplasma tend to be slow growing infections and they are usually transmitted slowly. Dr. Nicolson states that "Mycoplasma can be sexually transmitted, but the infection is usually passed through far less intimate contact. Mycoplasma can be obtained through fluid exchange, and it is easily transmitted through the air." In Gulf War veterans, the first person besides the veteran to become ill was the spouse and, later, other members of the household also became ill. Not everyone is equally susceptible to Mycoplasma infections, especially those with strong immune systems who can resist infection.
As already discussed, Mycoplasma fermentans produces numerous symptoms. Those infected are rarely found to be asymptomatic. In North America, M. pneumoniae is the most common Mycoplasma seen in various diseases. In Europe, M. hominis is far more prevalent and the incidence of M. fermentans is much lower than in North America.
The potential genetic factors involved in Mycoplasma illnesses are not known. Those with immune deficiencies and other illnesses, such as cancers and degenerative diseases, are at far greater risk of infection.

Prevalence

In one study looking at Mycoplasma in patients with Chronic Fatigue Syndrome, Dr. Nicolson has observed some interesting patterns in his research. Generally, the majority of CFS patients have Mycoplasma infections. However, CFS patients infected with Borrelia burgdorferi, the punitive agent in Lyme disease, had an even higher overall Mycoplasma infection rate. As many as 75% of Lyme disease patients appear to have Mycoplasma infections, and yet Mycoplasma is often overlooked in the diagnosis and treatment of chronic Lyme disease, neurodegenerative diseases, and many other chronic illnesses lacking clear origins.

Even more startling was the finding that of the patients infected with Borrelia, over 50% of the patients had the M. fermentans infection. Approximately 23% carried M. pneumoniae.

Chronic Fatigue patients that did not test positive for Borrelia had much more of a mixture of various species of Mycoplasma. Only 28% of the group not co-infected with Lyme disease had the M. fermentans infection. In normal, healthy controls, only 1.7% were found to have M. fermentans and at a total Mycoplasma infection rate of 5% compared to the 75% group mentioned earlier.

Dr. Nicolson notes that these findings are consistent with the fact that it is the Mycoplasma fermentans species that is more often isolated in ticks collected from the environment. The same tick that serves as the vector for Borrelia burgdorferi often also transmits M. fermentans simultaneously. Once a patient is multiply co-infected, the duration and severity of their illness both increase.

In his experience, Dr. Nicolson has found that Mycoplasma is the number one Lyme coinfection. The rate of infection with Mycoplasma in patients with Lyme disease surpasses that of Bartonella (25-40%) slightly and that of Babesia (8-20%) significantly.

According to Dr. Nicolson, a healthy immune system can generally clear M. pneumoniae infections though will have a harder time eradicating M. fermentans on its own. Healthy people can often hold these infections in check - essentially having the infection but not expressing symptoms.

Testing

Dr. Nicolson noted that Mycoplasma infections in chronic Lyme disease are often overlooked by most doctors because they simply don't test for it. He states that those that do test for it find a much higher number of infected patients. Dr. Richard Horowitz, MD in New York finds a high incidence of M. fermentans, according to Dr. Nicolson.

Sadly, however, even if patients are tested for Mycoplasma, a similar problem exists here as the one that almost all Lyme doctors and patients are aware of - namely that reliable tests do not exist. Dr. Nicolson notes that once a laboratory gets a reliable test in place, the laboratory is often shutdown. There are only a few labs left that test for Mycoplasma as a result.
In testing ticks for various microbial species, Dr. Nicolson has found a very high incidence of Mycoplasma fermentans. However, other Mycoplasma species have also been found such as M. pneumoniae and M. hominis. The incidence of these other species is far lower. "Far and away", it is the M. fermentans species that is seen in ticks, and this probably reflects the high incidence of M. fermentans coinfections in Lyme disease.

In terms of laboratory testing, Dr. Nicolson generally recommends Viral Immune Pathology, formerly known as RedLabs. He has found that the usefulness of any given lab in testing for Mycoplasma changes regularly. In the past, Dr. Nicolson used Medical Diagnostic Laboratories (MDL) for testing, but later he and other physicians found that the testing was no longer reliable. As a result, he no longer recommends MDL.
Dr. Nicolson finds that laboratories testing for Mycoplasma are highly scrutinized by federal agencies and that may affect the way the labs test and report this type of infection.

Autoimmunity

Thomas McPherson Brown, MD studied Mycoplasma at the Rockefeller Institute just before World War II. He was able to isolate bacteria from the joint fluid of a person with autoimmune arthritis and believed that the infection could have been the trigger for her disease. At the time, the organisms were too small to identify precisely, but it was later determined to be Mycoplasma.

Even then, Dr. Brown believed that Mycoplasma was very common and not easy to eradicate. He suggested using tetracycline drugs as an effective treatment for the disease. He later found that Doxycycline and Minocycline were effective at dealing with Mycoplasma. Though he garnered praise from his patients, he was generally regarded by the medical community as misguided and a trouble-maker. He died in 1989 prior to being fully vindicated. Fortunately, his work was validated through an NIH-sponsored study called MIRA or "Minocycline in Rheumatoid Arthritis".
Due to many of the characteristics of Mycoplasma, they may be responsible for the triggering of numerous autoimmune responses. As Mycoplasma replicate within cells and are eventually released, they capture antigens from the surface of the host cell and incorporate these antigens into their own membranes. This makes it almost impossible for the body to tell the difference between good and bad, between human and microbe, or between us and them. As a result, the immune system may begin to respond to these antigens now incorporated into the cell walls of the bacteria and create a condition of self-attack, or autoimmunity.

The microorganisms can produce mimicry antigens that mimic the natural host surface antigens and trigger an immune response to these antigens which may also result in autoimmune conditions through cross-reactivity. Additionally, Mycoplasma may cause cell death of host cells through a process known as apoptosis or programmed cell death.

Treatment

Though various strains of Mycoplasma have their own unique characteristics and drug responses, treatment tends to be quite similar. The variations in the strains do not appear to be a factor in a successful treatment response.

Dr. Nicolson suggests that in-vitro differences have been found but that it is not possible to easily extrapolate these findings to an in-vivo environment. Various factors including drug targeting, drug clearance, and the ability for the drug to cross into various body compartments are important considerations in treatment that cannot be examined in-vitro.
Dr. Nicolson believes that, like many other coinfections of Lyme disease, Mycoplasma cannot be fully eradicated, but that once infected, treatment becomes an ongoing "management approach". He notes that this is a commonly understood fact and that the same is true of other organisms such as Chlamydia and Borrelia. Mycoplasma have the ability to go into a quiescent phase in intracellular locations within the body. Once in these locations, neither antibiotics nor the immune system can effectively reach or kill the organisms.
Many people recover from Mycoplasma infections and are fine for years. They may later have an incident involving severe trauma or other significant life stressor and symptoms fully reappear within weeks to months.
Dr. Nicolson recommends that the physician adopt an initial 6-month course of treatment with no break followed by several 6-week on, 2-week off antibiotic cycles. Candidate antibiotics include: Doxycycline, Ciprofloxacin (Cipro), Azithromycin (Zithromax), Minocycline, or Clarithromycin (Biaxin). He notes that antibiotic combinations may be required if there is a limited response to single drug, and most patients require switching antibiotics at least once during their treatment. Some patients may find the addition of Flagyl to be a benefit to treatment.
In Gulf War patients, once effectively treated, the majority of patients recovered. For civilians, six months is the minimum recommended treatment length, and some patients require much longer treatment in order to recover.
Given that Mycoplasma have some characteristics of viruses, some physicians have suggested that Famvir or Ganciclovir may be added to the antibiotic therapy.

Herxheimer reactions do occur when treating Mycoplasma infections. To minimize this die-off effect where the patient generally feels much worse while on treatment, Dr. Nicolson advises using 50mg oral Benadryl taken 30 minutes before the antibiotics. He also finds that a strained blend of 1 whole lemon, 1 cup fruit juice, and 1 tablespoon of olive oil can be helpful.
Though Dr. Nicolson believes that antibiotics are the most effective approach to treating Mycoplasma infections, he has found some good natural options. In terms of natural approaches to treating Mycoplasma, Raintree Nutrition (http://www.rain-tree.com) has created several products that may be quite helpful for patients. These include Raintree Myco, Raintree A-F, and Raintree Immune Support.

Dr. Nicolson has seen evidence that Mycoplasma-specific transfer factors such as those from Chisholm Labs and others can be beneficial in some patients. He says that many natural options help in some patients, but that his experience has been that the antibiotic treatment results in the best outcomes. In many, recovery requires a push and pull between conventional and alternative treatments.

One of the hallmark signs of Mycoplasma infection is fatigue. The infections lead to oxidation in the body that leads to damage of the cell membranes. Oxidation accelerates the damage to the lipids in cell membranes which impacts mitochondrial function. This leads to less energy in the cell and ultimately to a fatiguing of the larger organism due to the fact that there is less energy to support necessary cellular functions.
In patients where fatigue is due to cell membrane damage, Dr. Nicolson has found NT Factor® to be highly beneficial. NT Factor® replaces the damaged lipids and helps to restore mitochondrial function. Often, fatigue then resolves or is reduced.
Dr. Nicolson has found that oxidative therapies such as ozone can be helpful in the fight against Mycoplasma. However, he notes that this is generally palliative and does not produce the same results as the antibiotic therapy in the long-term. He finds that the oxidative therapies "are generally more cytostatic than cytotoxic". Hyperbaric oxygen may be helpful but similarly does not appear to be a highly effective treatment in the longer-term.

In other countries, IV drips with H2O2 (hydrogen peroxide) have been used with some benefit, but Dr. Nicolson notes that these therapies, while potentially effective, are highly dangerous and not advised.
In the realm of frequency medicine and Rife therapy, Dr. Nicolson believes that the frequencies that could be used to address Mycoplasma are too similar to normal cellular frequencies. Thus, he is not certain that Rife therapy is an effective way to approach the problem.

In the nutritional realm, Dr. Nicolson finds that many patients with chronic infections are immunosuppressed and that proper nutrition is vital. He cautions against smoking and drinking. He suggests avoidance of sugars, trans-fats, and allergenic foods. He advises patients to increase their fruits, vegetables, and whole grains. Some dietary winners in supporting the immune system include cruciferous vegetables, soluble fiber-based foods such as prunes and bran, wheat germ, yogurt, fish, and whole grains.
Patients are often depleted in key vitamins and minerals. Supplementation with B-Complex, Vitamin C, Vitamin E, and CoQ-10 are often beneficial. Minerals are often necessary. Dr. Nicolson notes, however, that many people have poor absorption and may require sublingual or injectable forms of these nutrients. Amino acids, flax seed, and fish oils can provide additional support, but the best nutrition for cell membranes is NT Factor®.

Many patients with chronic illnesses have a toxic body burden of heavy metals such as mercury, lead, cadmium, and aluminum. Hair, stool, and urine testing is available through labs like Doctor's Data (http://www.doctorsdata.com) and Genova Diagnostics (http://www.gdx.net). Dr. Nicolson has seen reports of positive results with EDTA chelation suppositories from Detoxamin (http://www.detoxamin.com) and oral chelators from Longevity Plus (www.longevityplus.com).

For patients using antibiotics, beneficial gut flora is often depressed. Supplementation with a high quality probiotic is important, but probiotics have to be taken two hours or longer after taking antibiotics. Natural immune support can be helpful in the form of whey proteins, transfer factors, or immune-support products such as Beyond Immuni-T from Longevity Plus.

Biolfims

Dr. Nicolson believes that biofilms are a factor in successfully treating Mycoplasma infections. In cases that are refractory to antibiotics, biofilms are likely a major factor.

In men with chronic refractory prostatitis which is infection-based, one often cannot be treated effectively with antibiotics. However, when Detoxamin (EDTA) or other agents to address the biofilms are used, it then becomes possible to treat these infections with tetracyclines. Patients quickly show functional increases and decreases in pain other symptoms.

Summary

In chronic Lyme disease, it is often difficult to know which infections are actually responsible for the persistence of illness. However, in general terms, chronic intracellular infections that change the metabolism of cells and suppress mitochondrial and other functions will lead to patients remaining in a chronically ill state. Dr. Nicolson believes that these infections must be aggressively treated. "Similar to chronic Lyme disease, the current CDC or IDSA recommendations for short-term treatment of chronic infections are simply inadequate," he says.
Dr. Nicolson has found that there is a hierarchy of symptoms that resolve relatively quickly and those that resolve more slowly when treating Mycoplasma. Gut-associated phenomenon such as Irritable Bowel Syndrome (IBS) often resolve quickly. Other systemic signs and symptoms can resolve in an intermediate period of time from many weeks to many months. Symptoms associated with the central and peripheral nervous systems such as neuropathy and pain often resolve much more slowly. Skin sensitivity and burning sensations may take much longer to resolve. Mycoplasma infections do invade nerves, and nerve-related symptoms are among the more difficult to resolve.

Dr. Nicolson states "We keep seeing the suppression of information on Mycoplasma and similar intracellular bacterial infections. The world of Mycoplasma parallels the world of chronic Lyme disease in terms of the politics involved. Physicians are being persecuted by their medical boards as a result of bad information. It is important for us to do everything within our power to get rid of harmful, erroneous information about these diseases. Both Mycoplasma and Borrelia have been manipulated for biological weapons purposes and as a result, both are politically incorrect to discuss, work on, or do anything about. Until this changes, we won't see any real progress."

beautylicieuse.com/2012/01/pharmashopdiscount

2012-01-27T18:30:00.000+01:00

http://www.beautylicieuse.com/2012/01/pharmashopdiscount-vous-propose-de.html?utm_source=feedburner&utm_medium=feed&utm_campaign=Feed%3A+Beauty-licieuse+%28Beauty-Licieuse%29

La maladie de Lyme comme toutes les spirochétoses n’est pas une maladie infectieuse immunisante

2012-01-27T18:06:00.000+01:00

A.Boibieux
Maladies Infectieuses
Hôpital de la Croix Rousse
69004 Lyon
Exposé bref
9h00 Amphi 3

En France, la maladie de Lyme, zoonose transmise par la piqûre d’une tique du genre Ixodes,
est liée à une infection bactérienne par différentes espèces de Borrelia Burgdorferi sensu lato
(B. garinii, B. afzelii, B. Burgdorferi sensu stricto).
La primo-infection est exceptionnellement asymptomatique et se définit par l’érythème
migrant (EM). Non traitée, elle évolue vers le stade secondaire qui s’accompagne de
manifestations isolées ou associées, cutanées, articulaires, cardiaques, neuroméningées ou
oculaires. A la phase tertiaire, on retrouve des signes cutanés (acrodermite chronique
atrophiante), neurologiques, articulaires, musculaires et cardiaques.

Une primo-infection de l’adulte à B. burgdorferi se traite par 14 à 21 jours d’amoxicilline (3
g/jour), doxycycline (200 mg/jour), céfuroxime-axétil (500 mg x 2 fois/jour) ou 10 jours
d’azithromycine (500 mg/jour). Doxycycline est contre-indiquée chez l’enfant de moins de 8
ans et la femme enceinte.
Les molécules éligibles pour le traitement des phases secondaires ou tertiaires sont
amoxicilline, doxycycline et ceftriaxone (2 g/jour) pour une durée de 21 à 28 jours.
Dans certaine situation, une prolongation d’antibiothérapie peut-être proposée. Il n’est pas
recommandé d’assurer un suivi sérologique mais uniquement un suivi clinique.
La maladie de Lyme comme toutes les spirochétoses n’est pas une maladie infectieuse
immunisante. La protection mécanique repose sur le port de vêtements protecteurs longs et
fermés. Il faut retirer le plus vite possible la tique avec un tire-tique, désinfecter et surveiller la
zone de morsure. Une antibioprophylaxie systématique (dans les 72 heures après le
détachement de la tique) n’est pas recommandée mais discutée au par cas en cas
d’attachement de la tique depuis plus de 36 heures, en zone de forte endémie d’infection
des tiques (plus de 20%) et chez des patients immunodéprimés. Il n’y a pas de vaccin
disponible en France.
La morphée est le grand diagnostic différentiel de la maladie de Lyme et justifie d’une
biopsie de peau avec anatomie-pathologie et PCR Borrelia. Les infections à streptocoque et
staphylocoque sont plus connues après morsure par une tique que TIBOLA.
Les évolutions des écosystèmes imposent une surveillance attentive des zoonoses et tout
particulièrement de la maladie de Lyme.

Borréliose de Lyme et autres borrélioses. E.Pilly, 2010 : 309- 12.
Borréliose de Lyme, recommandations françaises (2006) :
‐ Texte court : http://www.infectiologie.com/site/medias/_documents/consensus/2006-
Lyme_court.pdf.
‐ Texte long : http://www.infectiologie.com/site/medias/_documents/consensus/2006-lymelong.
pdf.
Maladie de Lyme, recommandations américaines (2006 et 2010):
http://www.idsociety.org/lymedisease.htm.

http://www.lymedisease.org/news/lymepolicywonk/901.html

2012-01-27T18:00:00.002+01:00

http://www.lymedisease.org/news/lymepolicywonk/901.html

autre site parlant de la borreliose

2012-01-26T16:45:00.000+01:00



Bonjour a tous, je suppose que ce sujet sera encore longtemps actuel...malheureusement! Alors, je voulais apporter quelques infos concernant cette maladie... En Allemagne, il n'y a malheureusement aucune place pour cette maladie dans le milieu medical! a tel point qu'il m'a fallut moi-meme expliquer au medecin la multitude de symptomes possibles de la borreliose. Cette maladie ne sera surement pas acceptée avant longtemps par le milieu medicale dans la mesure où ses symptomes sont dans presque tout les domaines, comme Parkinson, sclerose en plaque, Tremblement essentiel familiale, infarctus, tintement d'oreille, vertige, tout les problemes articulaires, et - le meilleur pour la fin! alzheimer... Et j'en passe plein dautres. En fait, accepter tout ces symptomes comme venant de la borreliose serait une catastrophe economique pour l'industrie pharmaceutique! Pour les labo, c'est pire qu'en france : je n'en ai pas trouvé un qui soit competent dans le domaine et je me suis aussi tourné sur le labo de Viviane Schaller a strasbourg... La seul chance que nous avons en Allemagne vient d'un ethnologue-chamane - Wolf-dieter Storl, qui ayant eu la borreliose, l'a etudiée avec des moyens a la fois medicaux, ethonologique et chamanique... Une estimation allemande (parallele) dit qu'un allemand sur 7 aurait la borreliose! En effet, la borreliose n'est pas transmise par les tiques mais aussi par nous-meme! : par le sexe et par le sang. Comme pour le sida au debut, il n'y a pas de depistage de la borreliose pour les donneurs de sang! Donc a chaque transfusion, il y a risque de propagation!!! Curieusement, beaucoup d'individus développent une défense naturelle qui oblige les borrelies a s'encapsuler, etat dans lequel elle sont intouchables et peuvent rester 9 mois... Aucun antibiotique n'est donc efficace pour la meme simple raison qu'elles s'encapsulent aussitot... Selon Storl, ces bacteries sont les championnes de la survie. Elles seraient une variation plus evoluée de la syphilis... Il a donc regardé ce que faisaient les chamanes amerindiens avec la syphillis : ils modifiaient le terrain de la personne de telle facon que les bacteries ne pouvaient plus y vivre! En definitive - parce que je ne vais pas vous traduire le livre ici ;-) , il s'est gueri lui-meme par des frequentes sweat-lodges (les borrelies s'encapsulent au-dessus de 42°C) et une cure de cardere - plante qui modifie le terrain et le rend impropre aux borrelies... Cette cure de cardere a l'avantage d'etre preventive dans le cas de piqure de tique. En effet, les borrelies se trouvent dans l'estomac de la tique et analyse le sang que suce la tique. Si ce sang leur convient, elles migrent vers l'hote pour l'infester. Ce processus dure au moins dix-neuf heures. (donc pas de panique si vous voyez une tique le soir apres avoir fait une ballade en foret!). J'ai fait personnellement une cure de cardere de deux fois deux mois et une trentaine de sweat-lodges en un an et demi, mais les symptomes (surtout arthrite et tremblements des mains) demeurent... J'attends encore un peu pour aller ramasser des racines de cardere et me faire une nouvelle teinture-mere. Alors pour finir, comment faire une teinture-mere de racine de cardere : le cardere se développe en deux phase : la 1ere année, il fait une rosette au sol et rassemble toute son energie dans la racine (un rhizome) afin de pouvoir faire sa fleur l'année suivante. Donc c'est a l'automne a l'etat de rosette qu'il faut la ramasser, quand elle est pleine d'energie! Ensuite, on la rape et en bourre au maximum un pot etanche que l'on rempli d'alcool a 70%. Apres un mois de maceration, on extrait la teinture-mere en pressant les racines dans un linge de coton. Plus on presse fort, plus la teinture est concentrée. Il faut pas mal de cardere pour obtenir un kilo de racine mais cela depend ou elles poussent : ca peut aller du simple au decuple en poids! Personnellement je l'ai prise a raison de 30 gouttes avant chaque repas... J'imagine que l'ideal serait de faire une cure de plus de neuf mois pour etre certain que toutes sont mortes, mais je n'ai pas trouvé assez de carderes dans ma region!!! voilà, j'espere que ca peut en aider certains...

sur un autre blog

2012-01-26T15:06:00.000+01:00

La maladie de Lyme est véhiculée par les tiques. C'est une maladie peu connue, classée comme rare en France. En fait, elle n'est pas si rare que ça. Elle prend plusieurs formes et peu occasionner des troubles sévères. Il faudrait considérer la maladie de Lyme un peu comme la séropositivité et le sida, avec autant de sérieux. Malheureusement, par méconnaissance, beaucoup de médecins et même des hopitaux passent à coté pour leurs diagnostics. De très bon articles sont lisibles sur des sites spécialisés ou dans des actes de colloques.Cet article n'a pas pour objet d'inquiéter, mais d'apporter des éléments complémentaires aux informations déjà en circulation sur internet (par exemple sur francelyme.fr).

Si vous avez la maladie de Lyme (ou vous la suspectez), il y a des chances que votre foie soit fatigué. Vous devez alors l'épargner, en adaptant votre alimentation. Vous pouvez privilégier les légumes de saison, éviter l'alcool, le chocolat... ce qui vous alourdit lorsque vous mangez.

Par ailleurs, la maladie semble progresser surtout sur « terrain acide ». En fait, notre corps doit tendre vers un équilibre acido-basique, autour d'un ph de 7. Pour déterminer votre ph, des bandelettes sont en vente dans les magasins de diététique ou les pharmacies, avec des explications sur la façon de procéder et un code couleur pour déterminer votre ph. En gros, vous urinez et trempez la bandelette qui se colore (on privilégie la deuxième urine du matin, l'urine avant le repas de midi ou avant le repas du soir). Les tiques, comme els puces et les moustiques aiment les terrains acides, alors si vous agissez sur votre équilibre acido-basique, vous alliez préventif et curatif.

Pour gérer votre équilibre acido-basique, d'autant plus si vous avez Lyme, des changements de vie (stress, alimentation, repos...) risquent de s'imposer.

Un très bon livre de Christopher Vasey, « gérez votre équilibre acido-basique » aux éditions Jouvence est très clair et explicite.

Quelques petits trucs qui concernent Lyme:

Le dépistage: une analyse de sang classique ne suffit pas à déterminer si nous avons une forme ou une autre de maladie de Lyme. Il existe un laboratoire spécialisé dans les seules analyses fiables dans ce domaine, c'est le Laboratoire d'Analyse Médicales de Viviane SCHALLER, 16 rue Oberlin, 67000 STRASBOURG. Exigez de votre médecin une analyse dans ce laboratoire! Pour la sérologie de Lyme (test Elisa et Western Blot), vous ou votre médecin contacte le laboratoire qui envoie un kit pour le prélèvement de sang et toutes les instructions à suivre. Si votre médecin comprend que cette maladie n'est pas rare, il en commandera un carton (comme le font les médecins qui connaissent un peu cette maladie). En France, ce test est remboursé par la sécurité sociale.

Si vous habitez dans le sud de la France, comme moi, un docteur fait un travail formidable dans ce domaine, le Docteur DUGAUQUIER Sylvie à Saint Gaudens (05.61.89.43.79.).

Préventif: Si vous avez la maladie de Lyme, sachez qu'elle se transmet par le sang et le placenta. Aussi, merci de ne pas donner votre sang pour transfusion, car il n'y a aucun dépistage actuellement (un peu comme le sida à ses débuts...). Si après transfusion un ami à vous a des symptômes concernant Lyme, bien qu'il n'aie pas été piqué par une tique, un dépistage peut s'avérer aidant.

Préventif: Vous pouvez préparer un mélange préventif d'huiles essentielles (pelargonium, eucalyptus citronné, girofle, romarin, citronnelle). Ce mélange, j'en met 10 à 20 % dans de l'alcool (70 à 90 °), je laisse macérer puis je pulvérise su mes vêtements lorsque je vais en nature.

Ce même mélange, dilué dans une huile de cuisine (olive ou tournesol), je me l'applique sur la peau aux chevilles et poignets.

Curatif: chacun doit trouver sa propre voie. Certains utilisent l'homéopathie, d'autres la médecine chinoise, d'autres la naturopathie, d'autres l'antibiothérapie... Voici quelques clés concernant l'homéopathie:

En Allemagne, un remède majeur contre Lyme est la teinture mère de cardère, assez facile à faire.

En France, ceux qui utilisent la forme homéopathique de la borelia doivent se la procurer en Belgique, car la souche française est issue d'un vaccin alors que la souche belge est issue directement de la borelia. Pour la commander, une petite adresse mail: debrustensi@swing.be

Curatif: les borelia n'aiment pas la chaleur de type sauna. Alors si vous avez un sauna ou un hammam à proximlité, il est peut-être temps de prendre un abonnement.

Curatif: La vitamine C naturelle (acérola, argouses, mûres, cynorhodon...) sont des alliés précieux.

LES FORMES TARDIVES DE LA MALADIE DE LYME

2012-01-25T15:20:00.000+01:00

http://www.legeneraliste.fr/layout/Rub_FMC.cfm?espace=FMC&id_etiquette=M21&id_article=31745

LES FORMES TARDIVES DE LA MALADIE DE LYME

L’association de symptômes appartenant à des catégories différentes

Une maladie de Lyme non reconnue ou insuffisamment traitée à la phase primaire, risque d’évoluer vers une phase secondaire avec souvent un retard ou une absence de diagnostic liés à la grande variété de ses symptômes cliniques. Il faut avoir deux objectifs :
- identifier et traiter précocement l’affection au stade primaire pour éviter le passage aux autres stades
- penser à suspecter devant certaines symptomatologies « multi-organes », la possibilité d’une maladie de Lyme à un stade évolué.
La maladie de Lyme, due à Borrelia burgdorferi, est transmise par une piqûre de tique. En France, l’incidence moyenne est de 9,4 /100 000 (12 à 15 000 nouveaux cas par an) et entre 86 et 200/100 000 en Alsace (6). « Ces chiffres sont sous estimés, estime le Pr Perronne, la maladie est souvent non diagnostiquée et la sensibilité de la sérologie est médiocre.»

TRAITER VITE AU STADE PRIMAIRE.

La piqûre de tique passe inaperçue dans 69 % des cas. L’érythème migrant (EM), seul signe pathognomonique apparaissant 2 à 30 jours après la piqûre et disparaissant spontanément après quelques semaines, n’est présent que dans 70 à 80 % des cas. Et quand il est présent, il n’est pas toujours identifié ; soit que le patient ne consulte pas ou s’adresse seulement à la pharmacie, soit que EM n’est pas reconnu comme tel et étiqueté piqûre d’insecte, plaque d’allergie….
Aussi il importe de :
- éduquer ses patients à inspecter avec attention leurs corps après toute balade en forêt à la recherche de tique (sans oublier le cuir chevelu et les plis)
- enlever le plus tôt possible une tique, surveiller l’endroit pendant au moins 4 semaines et consulter dès l’apparition d’une rougeur (parfois d’un fébricule, de myalgies.) Pas d’antibiothérapie systématique sauf chez la femme enceinte (2).
- traiter toute personne présentant une lésion cutanée évocatrice d’EM (même sans notion de piqures) par une antibiothérapie suffisante (amoxicilline 3g/jour ou doxycycline 200mg/j pendant 2 à 3 semaines, sans attendre et sans faire de sérologie. (1). « Trop d’EM ne sont pas traités ou sont traités à des doses insuffisantes et/ou sur une durée trop courte, avec le risque de laisser la maladie de Lyme évoluer », déplore le Pr Perronne.

DES TABLEAUX CLINIQUES TRES VARIES

Non traitée au premier stade, la bactérie peut disséminer et être responsable de manifestations qui peuvent survenir quelques semaines à quelques mois plus tard. Ces symptômes possibles sont nombreux, divers, intermittents et extrêmement variables d’un sujet à l’autre. Aucun n’est spécifique, il peut s’agir de :
- asthénie intense, douleurs diffuses, parfois troubles de la mémoire et/ou de la concentration ;
- manifestations articulaires (arthralgies ou arthrites touchant une ou plusieurs articulations, les grosses surtout et le genou en particulier), musculaires ;
- manifestations neurologiques (céphalées, névralgies, radiculite souvent dans le territoire correspondant au lieu de la piqûre paralysie faciale à frigore, paresthésies, …), méningées ;
- manifestations cardiaques (tachycardie, extra-systoles, bradycardie,…), symptômes oculaires (conjonctivite, uvéite, troubles de la vision…),
- manifestations cutanées (morphée, rare lymphocytome cutané bénin) (4, 5).
Au stade tertiaire, qui évolue au fil des années si la maladie n’a pas été diagnostiquée et traitée, les mêmes catégories de symptômes deviennent chroniques, souvent permanents, parfois fluctuants.

QUAND EVOQUER UNE MALADIE DE LYME EVOLUEE ?

« Par la diversité de ses symptômes et leur absence de spécificité, la maladie de Lyme peut simuler beaucoup de maladies notamment auto-immunes. Penser à l’éventualité d’une origine infectieuse devant une symptomatologie floue et diverse permet, si elle est confirmée, de guérir ces patients ».
-› L’association de symptômes appartenant à des catégories différentes doit faire évoquer une maladie de Lyme, en exemple : une névralgie et une anomalie du rythme cardiaque, ou des paresthésies avec une asthénie et une arthralgie du genou, ou des signes oculaires avec des algies diffuses…
-› Ces patients ont habituellement un fond de fatigue et de douleurs mal expliquées et migratrices de type fibromyalgie, quelques paresthésies.
-› L’apparition de troubles des performances intellectuelles chez quelqu’un de jeune qui subitement présente des difficultés à se concentrer, à raisonner, des oublis est très évocatrice.
Il faut enquêter : ces patients, ont-ils déjà été piqués par une tique, se souviennent-ils d’avoir eu une plaque cutanée ? « Le premier obstacle au diagnostic, c’est que seulement 31 % des malades atteints se souviennent de la piqûre de tique. Ainsi, toute symptomatologie cutanée, oculaire, neurologique, méningée, psychiatrique, musculaire, cardiaque, osseuse, algique, etc… doit faire prescrire une sérologie de la maladie de Lyme, a fortiori si aucun autre diagnostic précis n’a pu être établi. »
Les stades secondaires ou tertiaires sont traités par antibiotique (amoxicilline 3gr/j ou doxycycline 200 mg/j) pendant 3 à 4 semaines.
En cas de forte suspicion clinique de maladie de Lyme (absence d’autre étiologie aux symptômes), si la sérologie est négative, un traitement antibiotique peut être proposé qui dans ce cas, a valeur de test thérapeutique.

Dr Catherine Freydt (rédactrice, fmc@legeneraliste.fr) sous la direction scientifique du Pr Christian Perronne (Chef du service de Médecine Aiguë Spécialisée, CHU Raymond Poincaré, 104 boulevard Raymond-Poincaré. 92380 Garches).

blog point beauté

2012-01-24T13:34:00.000+01:00

www.blogpointbeaute.com

nouveau concourrs

antibiotiques et lyme chronique

2012-01-14T16:46:00.000+01:00

http://www.plosone.org/article/info%3Adoi%2F10.1371%2Fjournal.pone.0029914

traitement de fin décembre à fin janvier 2011

2012-01-03T18:31:00.000+01:00

nouveau TMT

ORDIPHA 500 + doxy + tanakan +eurobiol

+ ...
le reste !!!!

blog de laura

2011-12-30T22:14:00.001+01:00

http://www.leblogdelaura.com/2011/12/concours-de-fin-dannee-mylo-filorga.html?showComment=1325278889357#c1079184578843929395

borréliose chronique et persistante

2011-12-21T23:55:00.000+01:00

http://www.dr-hopf-seidel.de/mediapool/87/874128/data/Persistierende_Borreliose_franz_Version_7-11.pdf

Lyme : quand demander une sérologie ? Pr Christian Perronne.

2011-12-20T17:54:00.001+01:00

Mardi 20 décembre 2011

Lyme : quand demander une sérologie ? Pr Christian Perronne.

Lyme : quand demander une sérologie
Les difficultés diagnostiques face à une suspicion de maladie de Lyme conduisent parfois à demander une sérologie, dont la sensibilité reste variable. Sa place n’est pas justifiée au stade primaire de la maladie, celui d’érythème migrant.

Piqûre de tique
La maladie de Lyme, due à Borrelia burgdorferi, est transmise par piqûre de tique, qui passe souvent inaperçue, d’autant que les larves et les lymphes de ces arthropodes peuvent être infestantes. La réduction de la pratique de la chasse qui a conduit à une prolifération du gibier, associée à l’augmentation de la pratique des loisirs de plein air sont à l’origine d’un accroissement de l’exposition de la population. On estime aujourd’hui l’incidence à 10/100 000 en France, certaines régions, comme l’Alsace où l’incidence est de 86/100 000, étant plus particulièrement concernées.
Pas de sérologie au stade primaire
Au cours de la phase primaire, le diagnostic est uniquement clinique : un érythème migrant au lieu de la piqûre est présent dans 70 à 80 % des cas. Il s’agit d’une lésion unique érythémateuse, survenant après une phase d’incubation de 3 à 32 jours, d’évolution centrifuge avec éclaircissement central très caractéristique. Le diamètre varie de 5 à 68 cm, avec une médiane de 15 cm, mais de véritables érythèmes migrants de taille inférieure à 5 cm sont possibles et alors bien souvent non diagnostiqués.
Au stade primaire, la recommandation consensuelle est de ne pas faire de sérologie, dont la négativité (80 % des cas) risquerait d’écarter faussement le diagnostic. En revanche, tout patient ayant une lésion évocatrice d’un érythème migrant doit bénéficier d’une antibiothérapie adaptée pour une durée de 14 à 21 jours et ce, même si la piqûre de tique est passée inaperçue. Il est essentiel de bien respecter la posologie (dose et durée) des antibiotiques, dont les deux principaux sont l’amoxicilline à raison de 3 g/jour et la doxycycline (200 mg/jour).
Chez un patient rapportant une histoire de piqûre de tique sans lésion cutanée, l’antibiothérapie n’est pas systématique, mais est par exemple indiquée chez la femme enceinte.
Phases ultérieures
De nombreux cas d’érythème migrant ne sont pas reconnus comme tels, ou sont insuffisamment traités (antibiothérapie trop courte ou à dose trop faible) et la maladie évolue alors vers le stade secondaire, où tous les organes peuvent être touchés, ce qui donne lieu à une grande variabilité d’expression clinique, puis tertiaire. Deux types de symptômes sont pathognomoniques de la maladie : le lymphocytome cutané bénin, lésion infiltrée inflammatoire généralement localisée au niveau du lobe de l’oreille, dont l’histologie est celle d’un lymphome. Et l’acrodermatite chronique atrophiante, vieillissement cutané prématuré le plus souvent au niveau des membres inférieurs, qui se rencontre fréquemment chez les sujets âgés.
En dehors de ces lésions pathognomoniques, le diagnostic est souvent difficile à faire à ces stades. En pratique, toute symptomatologie cutanée, oculaire, neurologique, psychiatrique, musculaire, cardiaque, osseuse… en l’absence de diagnostic précis, doit conduire à prescrire une sérologie de la maladie de Lyme, qui elle-même pose le problème de sa sensibilité.
En pratique
La recommandation officielle est de faire une sérologie en ELISA, IgM et IgG sans autre analyse en cas de négativité. En cas de positivité, le test doit alors être confirmé par un Western blot.
Le problème est que plusieurs études soulignent le manque de sensibilité des tests ELISA, qui varie de 37 à 70 %. En effet, l’impossibilité d’étalonner ces tests chez les patients conduit à réaliser l’étalonnage sur des sujets sains (donneurs de sang), la valeur seuil choisie visant à ne pas dépasser 5 % de séropositivité dans une région donnée. De ce fait, le seuil de positivité est arbitraire et varie d’une ville à une autre. Et c’est pour cette raison que sont régulièrement rapportés d’authentiques cas de maladie de Lyme (prouvés par culture de la bactérie) à sérologie négative.
Ceci a conduit les experts européens à recommander d’inclure dans les critères diagnostiques, en cas de forte suspicion de neuro-borréliose, la réponse à un traitement antibiotique d’épreuve.
Aux stades secondaires et tertiaires, l’antibiothérapie est prescrite pour une longue durée
› Dr ISABELLE HOPPENOT
Entretiens de Bichat. D’après la communication du Dr Christian Perronne, hôpital universitaire Raymond-Poincaré, Garches.
Le Quotidien du Médecin du 12/12/2011

Lyme Disease - My Story important, please watch!

2011-12-05T10:27:00.000+01:00

http://www.youtube.com/watch?v=em9OeiLkHUQ&feature=related

jeune américaine atteinte de lyme , dont les tests étaient négatifs puis positifs 2 ans après , qui vit un calvaire
écoutez son histoire , suivez ses conseils

la borreliose en europe

2011-12-03T15:30:00.001+01:00

http://www.youtube.com/watch?v=QxxpyGSZ4rU&feature=related

nouveau traitement

2011-12-03T15:06:00.001+01:00

j'ai commencé lundi mon nouveau traitement

c'est à dire 2,5 gr d'amoxi : L'amoxicilline est un antibiotique bêta-lactamine bactéricide de la famille des aminopénicillines indiqué dans le traitement des infections bactériennes à germes

et 200 mg de doxycycline ; la doxy me provoque souvent des herx , genre brûlures le long des nerfs craniens ; au moins ça veut dire qu'elle est efficace sur moi

Elles procèdent à la fois de l'activité antibactérienne et des propriétés pharmacocinétiques de la doxycycline. Elles tiennent compte à la fois de la situation de cet antibiotique dans l'éventail des produits antibactériens actuellement disponibles et des connaissances actualisées sur la résistance des espèces bactériennes.
- Elles sont limitées aux infections suivantes :
. brucellose,
. pasteurelloses,
. infections pulmonaires, génito-urinaires et ophtalmiques à Chlamydiae,
. infections pulmonaires, génito-urinaires à mycoplasmes,
. rickettsioses,
. Coxiella burnetii (fièvre Q),
. gonococcie,
. infections ORL et bronchopulmonaires à Haemophilus influenzae, en particulier exacerbations aiguës de bronchites chroniques,
. tréponèmes, (dans la syphilis, les tétracyclines ne sont indiquées qu'en cas d'allergie aux bêtalactamines),
. spirochètes (maladie de Lyme, leptospirose),
. choléra,

par contre j'ai perdu mon chien auquel je tenais beaucoup et je suis vraiment très triste
il ne faut pas de stress dans cette maladie mais j'ai passé toute la journée de mercredi à sangloter ma maman ayant été obligée de le faire euthanasier pour qu'il ne souffre plus ; j'ai énormément de chagrin
dimanche denier , il arrivait à marcher un tout petit peu seul , mais il tombait

tryptophan degradation in acute Lyme neuroborreliosis

2011-11-29T12:14:00.002+01:00

Eur J Clin Chem Clin Biochem. 1994 Sep;32(9):685-9.

Neopterin production and tryptophan degradation in acute Lyme neuroborreliosis versus late Lyme encephalopathy.

Gasse T, Murr C, Meyersbach P, Schmutzhard E, Wachter H, Fuchs D.

Source

Klinik für Neurologie, Universität Innsbruck, Austria.

Abstract

Fourteen patients with Borrelia burgdorferi infection were investigated for possible abnormalities of tryptophan and neopterin metabolism. Four patients (2 were investigated before therapy, 2 when therapy had been already started) had acute Lyme neuroborreliosis, and 10 patients were investigated months to years after an acute infection. Increased concentrations of neopterin and of the tryptophan-degradation product, L-kynurenine, were detected in the cerebrospinal fluid of patients with acute Lyme neuroborreliosis; one patient presented with subnormal tryptophan. Similar but less marked changes were seen in the treated patients and in some of the patients with Lyme encephalopathy. No such abnormalities were seen in the serum of the patients. The data indicate a role of the immune system and particularly of endogenously formed cytokines, like interferon-gamma and tumour necrosis factor-alpha, effecting tryptophan and neopterin metabolism in patients with acute Lyme neuroborreliosis.

PMID:: 7865624; [PubMed - indexed for MEDLINE]

Publication Types, MeSH Terms, Substances

LinkOut - more resources

traitement d'octobre 2011

2011-11-13T22:50:00.000+01:00

apres 15 jours de mosil , 1gr 6 /j + 750 mg de zinnat je trouve que la progression est immense , je n'avais pas eu ce traitement encore

je recommence à vivre , à marcher un peu parfois et j'ai plus le moral
je progresse incontestablement

une dizaine de jours d'arrêt et je repartirais avec doxy et amoxy

conférence 2011 Burrascano 2011

2011-11-08T11:36:00.000+01:00

Dr Joseph Burascano Conference du 21 Mars 2011 (notes)

Burrascano Notes from March 21st 2011 talk

Dr. Burrascano has treated over 10,000 patients with Lyme since early ‘80s.

“It is up to you. Learn as much as possible. Do as much as possible. Have a positive attitude.”

Dr. Burrascano’s definition of Lyme Disease
“Lyme disease is the illness that results from the bite of an infected deer tick; it’s not one germ.”

Stages of Lyme

Early Lyme – I
Disseminated Lyme – II
Chronic Lyme – III
-for one or more years – immune system breakdown and its consequences
-co-infections become important
-serologic tests less reliable
-treatment must be more aggressive and of longer duration

The sicker you are, the less reliable the tests; lyme burrows deeper and is no longer easily detected in blood

CD-57 test – the one test that shows how long Lyme has been present (See more blow regarding CD57)

Tick Bites

Only 17% recall having a tick bite (Texas Dept of Health)
Only 36% recall a rash
Only 50% have positive western blot
Co-infections: tests are even less sensitive

Ticks: nature’s “dirty needles”; a tick lives for 2 years

Co-infected patients: more ill, more difficult to treat; Lyme treatments do not treat Babesia, Bartonella, or viruses.

**Dr. Burrascano says he has never seen a patient without co-infections.

Sorting Out Co-Infections
Lyme, Bartonella, Babesia, Ehrlichia, Mycoplamsa

Lyme – Gradual onset, no sweats, 4 week cycles, multisystem, afternoon fevers

It is important to take your temperature several times a day (record in journal)

Babesia – Cycles every few days, makes everything worse

Ehrlichia – Sharp headaches behind eyes, low WBC, elevated liver function

Mycoplasma – Made worse with exercise, aka “Chronic Fatigue Germ”, major fatigue, neurological disfunction, found in the sickest and poorest responding; have the worst CD-57 tests

ELISA test – mostly useless; use Western Blot
Spinal Tap – only 9% have + csf
PCRs – 60% sensitivity at best because germ doesn’t stay in blood
LDA – 30% sensitivity

Why Igenex?

Dr. B has no affiliation with them, no professional relationship with them, etc…

If a test is commercialized to be sold as a test kit, it must follow narrow CDC restrictions and guidelines. (Ironically, these restrictions were a result of the Lyme vaccine debacle.)

Most Lyme tests are commercial. Commercial Lyme tests miss 75% of cases.

Based on double-blind government proficiency tests, IGenex did well.

CD-57 COUNT (tracks a type of white blood cell)

Lower counts seen in chronic Lyme
Only Lyme (not co-infections) makes CD-57 low

The CD-57 reading does not change *during* treatment … until Bb is controlled. Then it quickly changes.

Predicts a relapse if low when antibiotics end

The CD-57 test must be done by LabCorp’s method (using the “normal is >200” scale)
60 Lyme activity minimal
> 120 – relapse not likely

Why Are Chronic Lyme Patients So Sick?

-High spirochete load (perhaps multiple bites)
-Protective niches in the body and biofilms allow Bb to evade the immune system and antibiotics
-Immune suppression and immune evasion

Biofilms are a protective layer

Lyme germs live in different forms:
Spirochete – surrounded by a cell wall
Spiroplast – balls up, has no cell wall
Cystic form – has hard outer shell

Lyme germs can live *inside* a human cell, inside the vacule

Doxycycline – can get into the cell
Erithromycin – can get into the cell
Rocephin – does not kill germs inside vacule

Treatment - Back to Basics

Form a therapeutic alliance with your Dr.; should be able to have “meeting of the minds”

Pharmacology
-It is *critical* that you achieve therapeutic drug levels – this varies from patient to patient
-Antibiotics – you *must* have extra-cellular and intra-cellular meds as Bb can live in and out of cells
-Antobiotics – must act on blood & tissues

Spirochete forms:
Penicillins, Cephalosporins, Primaxin, Vancomycin,

Spiroplast/L form: no cell wall
Tetracyclines, Erythromycin

Cyst:
Metronidazole, Tinidazole, Rifampin

Spirochete B. burgdorferi – needs sustained levels
L form – Tetracyclines, need a spike in blood levels
Cystic – Metronidazole, sustained levels for 2 weeks +

Antibiotic combinations are necessary
Intracellular and extracellular
Blood and tissue

Intravenous therapy is most effective
Intramuscular Penicillin effective as well

Indications For Intravenous Therapy
-illness for more than one year
-prior use of steroids
-documented immune deficiency
-abnormal spinal fluid
-synovitis with high ESR
-age over 60
-failure or intolerance of oral therapy

Typical Regimen

Oral
Cefuroxime + Clarithromycin
Augmentin XR + Telithromycin

Injection
BicillinLA + Clarithromycin

Intravenous
Clarithromycin + Telithromycin
Vancomycin + Clarithromycin

-high doses needed
-combination usually necessary
-check for co-infections
-rotate treatments

Rate of recovery dependent on germ; stronger drug will not speed recovery.

Find a regimen that works and stick with it
Change when you’ve reached a plateau
Treatments: at least 4-6 weeks before changes

Relapses

-relapses occur; retreatment needed
-repeated and/or prolonged antibiotic therapy

Aggressive supportive therapy also necessary:
Sleep cycle
Food
Supplements
Detoxing

As symptoms wind down, DO NOT cut dosage! Resistance develops that way.

Progressively increase exercise program
-exercise is vital and required
-not exercising will increase risk of relapse

If CD-57 is not normal at end of treatment, continue treatment or there will be relapse

Prognosis
-May not cure infections, may need open-ended maintenance therapy

What to Watch For:
Signs of persistence; continued fevers
Four week cycles of ailments
Migrating symptoms
Positive PCR or urine LDA

If you have not relapsed in 3 years, you never will.

What if you’re not sure you’re over it?
Low grade fever still present
Signs of recurrent four-week cycles
Migrating pain
Low CD-57 counts

Bartonella
The Bartonella co-infection with Lyme seems to be clinically different that “cat scratch”.
Instead, they are Bartonella-like organisms; more prevelent that Borrelia in some ticks

Clinical Clues
Encephalopathy
Irritability
Anxiety
Stomach lining
Insomnia
Rashes
AM fevers
Night sweats
Tender skin nodules

Bartonella treatment:
Levaquin Fluoroquinolones

Erithromycins don’t kill this
Rifampin & Metronidazole may be alternatives
1 – 3 months of treatment

Piroplasms – Babesia
Is a parasite

Symptoms
Night sweats
Air hunger
An occasional cough
Persistent migraine-like headache
A vague sense of imbalance without true vertigo
Encephalopathy
Fatigue

Babesia Treatment
Not treated with antiobiotics

Azithromycine & Mepron
Malarone
Coartem – Antimalarial for Babesia (new)

Ehrlichia
Headaches
Muscle soreness
Persistent leucopenia (low WBC)

Treatment
Doxycycline 1st choice

Mycoplasms
“Chronic fatigue” germ
Ubiquitious in environment (in dust, for example)
Treatment is difficult

Worms
New species of nematodes in 63 – 75% of patients from Massachusetts

Fatigue
Lives in lungs mainly
(Dr. Eva Sapi, Dr. Larry Klapow – research)

An open mind is important!!

Biofilms
Dental plaque is an example
Gel-like substance in which germs can embed
Biolfilms in the gut are implicated in many digestive diseases & possibly food allergies and mal-absorption

Biofilm busters:
Banderol plus Samento
Enzymes

Methylation Cycle
Key component of metabolism
Need to bring up methylation cycle
This cycle can be blocked when chronically ill
75% of Lyme patients responded better after treating

Crazy or Is It Lyme?
Cytokines – mediators of inflammation, are activated.
When this occurs in the CNS, it triggers diversion of tryptophan into kynurenine
Result: depression, neuropathy, fog brain, “crazy” perception

Bornavirus
Autism-related?
Distant cousin to Rabies and distemper
Brain is the site of infection
Does not damage nerve cells but blocks cell function
Brain fog, fatigue leads to depression

Treatment:
Antiviral Amantadin
65 – 70% success rate

XMRV
Xenotropic Murine Leukemia Virus
Virus – is gamaretrovirus, 1st isolated in prostate cancer
68 out of 101 CFIDS samples contained XMRV
XMRV is found on only 3% of healthy samples
XMRV is also found present in cases of: MS, ALS, Parkinsons, Autism, Fibromyalgia
Lyme patients who did not recover: 1005 of them had XMRV

Does it prevent a full recovery from Lyme?
Is it found in ticks?

Dr. Judy Mikovits

Treatment – XMRV
Retrovirus is cousin to HIV
-be sure there is not excessive cortisol and DHT
-consider adding antivirals AZT, tenofovir, raltegravir

Basic Advice

NO steroids or other immune suppressives!
No smoking at all
No alcohol (makes germs stronger, weakens immune system)
Clean diet: low carb, low glycemic index, high quality proteins
Maintain hydration (Lyme patients become dehydrated quickly, sense of thirst is altered)
May need mineral supplements

ENFORCED REST

You are NOT allowed to get tired
Take a break before afternoon lag
Work and school – Go in later, leave earlier, take a midday break, take Wednesdays off
Rest on days off
No caffeine, no stimulants
Home should be quiet, comfortable, non-toxic
Nap if needed!!
If you need to sleep late, do it!!

Exercise Program

Body sculpting
Gentle with free weights, exercising all muscles; very light or no weights
NO AEROBICS
Each body sculpting session 45 min; 60 minutes preferred
Begin with good progressive warm-up
Take a hot shower or bath afterward and go to bed. Lie quietly if you cannot sleep.
Never exercise daily
Total rest on off days
As strength improves, increase weight and resistance but maintain high number of repetitions
As stamina improves, exercise more, but NEVER daily.

Dr. B believes: spirochetes choose skin as their final hiding place.

Nutritional Supplements

Probiotics

Multivitamins w/minerals

CoQ10 or ubiquinone

NT-factor or “ATP fuel”

ATP Fuel

Vitamin D – maintain upper-normal levels

Essential Fatty Acids

Magnesium

Methyl B-12

B complex

Transfer Factors

Detoxify

FIR Saunas: Helpful to excrete organic toxins

PLEASE NOTE: Because the presentation was so dense and rich with information, there may be some info that was left out.

DEET does not work!! Use Premethrin!

(Developed in cooperation with the U.S. Military, government agencies, universities and others; this Sawyer Clothing repellent offers superior protection from disease-carrying biting insects. The active ingredient, Permethrin is a synthetic molecule similar to those found in natural pyrethrum which is taken from the Chrysanthemum flower. Not only does this product repel insects, but will actually kill ticks, mosquitoes, chiggers, mites and more than 55 other kinds of insects. Sawyer Permethrin repellent is for use on your clothing, tents and other gear. A single application lasts up to six weeks and will remain effective even if you wash the garment once a week. Permethrin is odorless when dry, and during the drying process it tightly bonds with the fibers of the treated garment, it will not stain or damage clothing, fabrics, plastics, finished surfaces, or any of your outdoor gear. Try our two easy-to-use treatment methods: aerosol spray and NEW soak system. Both methods provide protection from mosquitoes and ticks through 6 launderings.)

And the most important part.......

Live with a healthy attitude;
Lose “poor me”, lose anger

Do not become “Lyme obsessed”
Pursue other interests and distractions
Enjoy friends and family
Cuddle with your pets

I want to thank you my friend for taking these wonderful notes for me and for all of you!

Labels: Burrascano, CALDA, Lyme Disease, March 21, Santa Rosa

spirochetesunwound

2011-11-04T17:59:00.003+01:00

http://spirochetesunwound.blogspot.com/2011/10/tale-of-two-more-studies-topical.html

Sunday, October 30, 2011

A tale of two more studies: topical antibiotics applied to tick bites to prevent Lyme disease

Feeding Ixodes ticks harboring Borrelia burgdorferi deposit the Lyme disease spirochete in the skin of the victim. The spirochetes remain in the skin for a few days before entering the bloodstream to spread throughout the host. The delay in dissemination provides a window of opportunity to stop the infection by simply applying antibiotics to the skin where the tick was feeding. Topical application of antibiotics would allow patients to avoid experiencing side effects associated with ingesting antibiotics.

I recently posted a critique of a study by Knauer and colleagues, who tested the ability of a topical antibiotic to prevent B. burgdorferi infection in lab mice bitten by infected ticks. As I explained in the post, the antibiotic appeared to prevent infection, but the investigators had used a weakened B. burgdorferi strain to inoculate the mice. Consequently it wasn't possible to draw any conclusions about the effectiveness of their antibiotic formulation in preventing infection.

Now let's look at two more studies that tested the ability of topical antibiotics to prevent infection in the mouse model of Lyme disease. These studies were conducted properly with highly infectious B. burgdorferi strains. One study was published almost 20 years ago. The other appeared online just last month. Both studies were published in The Journal of Infectious Diseases.

In their 1993 study Shih and Spielman were able to prevent B. burgdorferi infection by applying at least 1 milligram of tetracycline starting up to two days following the tick bite. The antibiotic had to be applied twice a day for at least three consecutive days (see table below). The presence of infection four weeks after tick feeding was assessed by xenodiagnosis, which tests whether the spirochetes could be recovered by ticks placed on the skin at a location distant from the original tick bite. The investigators also found that penicillin, amoxicillin, ceftriaxone, doxycycline, and erythromycin applied for three days beginning one day after tick feeding prevented infection.

Although this study demonstrated the promise of topical antibiotics in preventing Lyme disease in those who discover a tick feeding on them, a limitation of the study was that most of the antibiotics were dissolved in DMSO, which is not approved for use on humans. The only antibiotic dissolved in a solvent suitable for humans was erythromycin, which was added into 70% ethanol.

For their 2011 study, Wormser and colleagues decided to dissolve the antibiotics in something else that would be acceptable to apply to human skin. They rubbed a 2% erythromycin ointment or 3% tetracycline gel over the tick bite 1-2 days after the infected ticks finished feeding on the mice. The antibiotics were applied twice daily for three days. Four weeks later urinary bladder and ear tissue were cultured to see whether the mice had a disseminated infection. The authors found that their antibiotic formulations failed to prevent systemic infection, although erythromycin was able to prevent a persistent infection at the tick bite site in some of the mice (see table below).

Erythromycin and tetracycline were tested in both studies. Why the stark difference in the effectiveness of the same antibiotics in the two studies? In the Discussion of their paper, Wormser and colleagues highlighted the major methodological differences between the studies:

Different antibiotic concentrations. A much higher concentration of erythromycin was applied to the tick bites in the 1993 study.
Different solvents. DMSO, the solvent used for the 1993 study, may have promoted better penetration of tetracycline into the skin than the ointment and gel formulations selected for the Wormser study.
Different placement of infected ticks. In the 1993 study the infected ticks were placed on the ear for feeding. In the Wormser study the ticks were placed on the back, where the skin may be thicker and hence more resistant to antibiotic penetration.
Different B. burgdorferi strains. Wormser and colleagues used ticks infected with the highly invasive BL206 strain to inoculate the mice, whereas Shih and Spielman used ticks infected with the less invasive JD1 strain.
Different mouse strains. The C3H mouse strain used in the Wormser study is highly susceptible to dissemination by B. burgdorferi.

For this simple treatment approach to effective, higher concentrations of the antibiotic in a penetrating solvent such as ethanol may be necessary. Different B. burgdorferi and mouse strains should also be tested in future studies.

References

Shih, C.-M., & Spielman, A. (1993). Topical prophylaxis for Lyme disease after tick bite in a rodent model Journal of Infectious Diseases, 168 (4), 1042-1045 DOI: 10.1093/infdis/168.4.1042

Wormser, G.P., Daniels, T.J., Bittker, S., Cooper, D., Wang, G., & Pavia, C.S. (2011). Failure of topical antibiotics to prevent disseminated Borrelia burgdorferi infection following a tick bite in C3H/HeJ mice Journal of Infectious Diseases DOI: 10.1093/infdis/jir382
Knauer, J., Krupka, I., Fueldner, C., Lehmann, J., & Straubinger, R.K. (2011). Evaluation of the preventive capacities of a topically applied azithromycin formulation against Lyme borreliosis in a murine model Journal of Antimicrobial Chemotherapy DOI: 10.1093/jac/dkr371

Related post

A flawed study claiming prevention of Lyme spirochete infection with topical antibiotics

Direct0bio : j'ai testé pour vous le Saint Domingue Ocoas

2011-11-04T16:34:00.002+01:00

le Saint Domingue Ocoas !

voici mon commentaire sur ce café : tout d'abord l'odeur qui passe à travers le paquet est absolument divine , elle donne tout de suite envie de goûter à ce café, malheureusement mon paquet était un peu coupé sur le côté ce qui fait qu'un peu de café s'échappait, mais ce n'était pas grave

la mouture n'est pas fine , elle ressemble à celle que l'on obtenait dans les anciens moulins à café , surtout quand on moulinait les grains à la main , ce qui nous a surpris ;
puis est venu le moment de la dégustation :
franchement il y a vraiment tres longtemps que je n'avais bu un aussi bon café filtre , un café qui a du goût , aucune amertume , un café qui reste bien en bouche et dont l'odeur vous titille les narines ; nous le recommandons fortement , vous ne serez pas déçus , il est grandiose ;
merci à Direct0bio de nous avoir fait tester le Saint Domingue Ocoas , il nous a fait rêver, je le recommande fortement à tous les amateurs de bon café

http://www.directobio.com/cafe-saint-domingue-ocoas-moulu-filtre-250g-p-337-1_40.html

maladie psychiatrique dues à des réactions auto-immunitaires

2011-11-04T00:39:00.000+01:00

Défensif et offensif

> Le système immunitaire peut, par erreur, endommager le cerveau d'une autre façon (et pas seulement chez le fœtus).
> Même si les preuves scientifiques indiquent un lien entre les maladies mentales et les infections prénatales, beaucoup de recherches sont faites afin de voir s'il peut y avoir des cas de maladie psychiatrique dues à des réactions auto-immunitaires déclenchées lors de l'enfance ou même de l'âge adulte.
> Certaines infections peuvent déclencher le système immunitaire et lui faire attaquer des cellules du cerveau.
> Une de ces infections est une infection due aux streptocoques (ceux qui sont responsables de l'angine rouge).
> En 1998, les médecins, l'équipe du docteur Swedo, faisaient une étude sur les enfants qui souffraient de Troubles Obsessionnels du Comportement (TOC) et constatèrent qu'un petit pourcentage de ces enfants avaient tout d'un coup développé ces TOCs après une infection due au streptocoque bêta hémolytique du groupe A.
> Généralement, les TOC viennent progressivement, mais chez ces enfants, on était passé d’aucun symptôme à TOC en l'espace de 24 ou 36 heures.
> En gros, ces enfants se sont réveillés un matin et avaient des TOCs.
> C'est pour cela que les TOCs dus à cette condition, aux États-Unis, ont une dénomination bien particulière : PANDAS (Pediatric Autoimmune Neuropsychiatric Disorders Associated with Streptococcal infections).
> D'après le docteur Swedo et son équipe, PANDAS se développent, car le système immunitaire attaque le cerveau après l'infection.
> La bactérie du Streptoccoque contient certaines protéines à sa surface qui ressemble aux protéines faites par les humains. C'est ce qui aide la bactérie à ne pas se faire éjecter par son hôte.
> Lorsque le corps découvre la bactérie, il l'attaque, et dans ce processus de défense, il attaque aussi ses propres protéines.
> Certaines études ont trouvé des anticorps contre le cerveau chez des patients atteints du PANDAS, mais d'autres études n'ont pas pu reproduire l'expérience.
> Ce qui fait que le diagnostic du PANDAS est toujours sujet de controverse.
> Mais même si on a toujours certains doutes, la plupart des scientifiques s'accordent à penser que le PANDAS est une pièce importante du puzzle.
> Si la raison est une réaction auto-immune ou une interruption du développement du cerveau foetal, si le coupable est le système immunitaire plutôt que l'infection en elle-même, ceci simplifie grandement le problème.
> Et cela nous expliquerait pourquoi on trouve plusieurs maladies impliquées dans les maladies psychiatriques : grippe, rubéole, herpes, etc.
> Ces infections sont très différentes et agissent différemment sur notre corps, oui, mais le point commun est notre système immunitaire.
> Les chercheurs espèrent pouvoir découvrir les raisons des maladies mentales et espèrent un jour trouver une solution, une cure.
> Le plus important, si on veut combattre les maladies mentales, est de les éviter au premier abord. Si les infections jouent un rôle crucial, cela ouvre les portes pour de nombreuses solutions.
> Mais cela pourrait aussi changer certaines des habitudes que nous avons, car si, comme tout porte à le croire en ce moment, c'est le système immunitaire, et non l'infection en elle-même qui affecte le fœtus, il va falloir arrêter de conseiller aux femmes enceintes de se faire vacciner contre la grippe.
> Peut-être arrivera-t-on à des médicaments qui viseront directement l'infection ou qui contrôleront l'interférence du système immunitaire avec le cerveau, afin d'éviter une attaque auto-immunitaire sur les cellules du cerveau.
> Le 20e siècle a curé toute une série de maladies physiques dues à des infections, peut-être que le 21e siècle verra une cure aux infections qui affectent notre cerveau ?

Des études récentes indiquent une grande variété de liens entre certaines infections et des éléments psychiatriques. Voici certaines des corréaltions les mieux documentées à l'heure actuelle.

Schizophrénie	Prénatal	Grippe, rubéole, Taxoplasma gondii, herpes, polio, varicelle, maladie de lyme.
Schizophrénie	Postnatal	Taxoplasma gondii, maladie de lyme, chlamydia, herpes.
TOC	Prénatal	Aucun lien trouvé.
TOC	Postnatal	Streptocoques.
Bipolaire	Prénatal	Herpes, Taxoplasma gondii.
Bipolaire	Postnatal	Herpes, Taxoplasma gondii.
Autisme	Prénatal	Rubéole, herpes, maladie de lyme.
Autisme	Postnatal	Maladie de lyme, mycoplasmose, clostridium (bactérie qui cause le botulisme).
Alzheimer	Prénatal	Herpes.
Alzheimer	Postnatal	Aucun lien trouvé.
> Syndrome de Tourette	Prénatal	Aucun lien trouvé.
> Syndrome de Tourette	Postnatal	Mycoplasme.

> (Cyril Malka d'après Melinda Wenner)

Pr Montagnier : recherches sur lyme

2011-11-03T00:23:00.001+01:00

http://83.143.18.11/sante-et-forme/videos/avancee-de-la-recherche-sur-la-maladie-de-lyme-par-le-pr-luc-montagnier.html

Anne Ghesquière, fondatrice de FemininBio.com a rencontré le Professeur Luc Montagnier Président de la Fondation Mondiale Recherche et Prévention SIDA.

Le Pr Luc Montagnier s'exprime en particulier sur la maladie de Lyme, mais aussi sur d'autres thèmes comme notre système immunitaire, le stress oxydant (cause du vieillissement de nos cellules), les avancées dans la recherche sur le SIDA et l'eau. Le lien entre tous ces thèmes est bien entendu "le terrain" et donc la réponse de notre système immunitaire.

http://83.143.18.11/sante-et-forme/videos/avancee-de-la-recherche-sur-la-maladie-de-lyme-par-le-pr-luc-montagnier.html

droopyyoupi

Blogiversaire jour 1

un médecin traitant lyme se voit "mis à pied "

les mycoplasmes dans la maladie de lyme chronique

beautylicieuse.com/2012/01/pharmashopdiscount

La maladie de Lyme comme toutes les spirochétoses n’est pas une maladie infectieuse immunisante

http://www.lymedisease.org/news/lymepolicywonk/901.html

autre site parlant de la borreliose

sur un autre blog

LES FORMES TARDIVES DE LA MALADIE DE LYME

LES FORMES TARDIVES DE LA MALADIE DE LYME

blog point beauté

antibiotiques et lyme chronique

traitement de fin décembre à fin janvier 2011

blog de laura

borréliose chronique et persistante

Lyme : quand demander une sérologie ? Pr Christian Perronne.

Lyme : quand demander une sérologie ? Pr Christian Perronne.

Lyme Disease - My Story *important, please watch!*

la borreliose en europe

nouveau traitement

tryptophan degradation in acute Lyme neuroborreliosis

Neopterin production and tryptophan degradation in acute Lyme neuroborreliosis versus late Lyme encephalopathy.

Source

Abstract

Publication Types, MeSH Terms, Substances

LinkOut - more resources

traitement d'octobre 2011

conférence 2011 Burrascano 2011

Dr Joseph Burascano Conference du 21 Mars 2011 (notes)

spirochetesunwound

Sunday, October 30, 2011

A tale of two more studies: topical antibiotics applied to tick bites to prevent Lyme disease

Direct0bio : j'ai testé pour vous le Saint Domingue Ocoas

le Saint Domingue Ocoas !

maladie psychiatrique dues à des réactions auto-immunitaires

Pr Montagnier : recherches sur lyme

Lyme Disease - My Story important, please watch!