Anuradha Karthikeyan

SUPER STAR

2012-01-22T06:27:00.001-08:00

This is not to become one among the Rajinikanth jokes, but it's real. A website has been launched to honor, larger than-life, Indian film superstar that runs without an internet connection. Yes. You read it right.

www.allaboutrajni.com

ARUNA SAIRAM

2011-03-22T08:50:00.000-07:00

Baje Mridunga

Kalinga nartanam

VishamaKaara Kannan

Maadu Meikum Kanne

Brindavane

RAAKU KAALAM

2010-12-15T23:26:00.000-08:00

ராகு காலம்

How to remember Rahu Kalam

Although the time Rahukaalam falls on each day, and its span varies and needs to be calculated as per the sunrise and sunset time. You may use the times below if you do not have access to accurate Rahukaal.

These times are accurate only if the day lasts for 12 hours, and the sunrise is at 6.00 AM. You can adjust an approximation according to the sunrise and day length in your area.

All you need to remember is the phrase " [Mother Saw Father Wearing The Turban Slowly]"

Mother = Monday : 7:30 - 9:00
Saw = Saturday : 9:00 - 10:30
Father = Friday : 10:30 - 12:00
Wearing = Wednesday : 12:00 - 13:30
The = Thursday : 13:30 - 15:00
Turban = Tuesday : 15:00 - 16:30
Slowly = Sunday : 16:30 - 18:00

Instead of the above one there's another easy way to remember: " [--Edward Brought Home A Good Ford car.]"
Starting with Monday count the First letter of every word and add half of the same which is 11/2 Ex: E is the letter 5+21/2 = 7:30 so Monday its 7:30- 9:00.

(Edward) Monday - 7:30 -9:00

(Brought) Tuesday - 3:00 -4:30PM

(Home) Wednesday - 12 - 1:30PM

(A) Thursday - 1:30 - 3:00PM

(Good) Friday - 10:30 - 12:00

(Ford) Saturday - 9:00 - 10:30

(car) Sunday - 4:30 - 6:30PM

Eleven Boys Had A Good Football Club is a very popular way as above to remember Rahu Kalam duration.

http://en.wikipedia.org/wiki/Rahukaalam

My garden

2009-11-29T22:16:00.001-08:00

Sri mathura kali amman

2009-11-29T22:02:00.000-08:00

Orange County

2009-11-28T07:49:00.001-08:00

Coorg is a beautiful hill station in the western ghats. Whichever side you turn the nature blesses you with bountiful greenery and thick, dense forested mountains. In the midst of such bliss lies the famous orange county resorts. As the name suggests, this place had once been full of orange trees. But now you can find coffee plantations with a beautiful fragrance of the flowers of coffee plants. It also carries a beautiful combination of rubber trees along with pepper climbers.

The river Cauvery, which originates from the Talaikaveri flows through Orange county. This adds glamour to the whole mountain. We have boating facilities and the boat rushes into the river which is nearly 70 feet deep. And so we are cautioned to wear life jackets. The boatman takes us to a place where the river is a bit shallow so that we can a take a dip in the river. Both the banks of river Cauvery are filled with tall trees and a good fertile vegetation. We can have a sight of rare birds with beautiful feathers and a good number of animal population.

Rare sights of elephants and tigers are reported. A number of white feathered peacocks, turkies, swans, storks, pegions etc. surround us at once we come out of the cottage.

The cottage, especially the thatched cottages we choose will make us live with the nature. The cottage will have a very comfortable bed with a balcony. The toilet rooms are neatly furnished. Coffee powder, sweetener, milk powder, hot water will be readily available in the room. A morning walk around the orange county will refresh us just like anything. The freshness of the grass left behind the dew drops will surely brighten our eyes.

Nights are extremely cold in orange county especially in the season from november to february. The clear dark skies with twinkling stars simply speak to us. Jerkins, socks and sweaters are a must in this season.

The food provided in the orange county restaurant is simply divine. we will get all type of food and there are certain favourite dishes of mine.
1) sugarcane juice.
2) appam with vegetable stew.
3) coffee.

The chillness of the weather and the moisture content in the air rejuvenates our nerve and brain cells.

Moreover it has an ayurvedic center with all types of massage, a recreation hall with games like table tennis, carrom, chess etc., an anthurium garden beside a natural lake within orange county, an artificially created lake where boating will permitted in off season(this is in addition to boating in river cauvery), an antique gift shop etc.

Besides these we can visit thalaikaveri if we hire a cab.

Such a divine place always is a heaven for honeymooners. they can thoroughly enjoy here.

How to go?
Orange County is just three hours from bangalore.

Bookings and other enquiries
Contact
08041911044
04442104541

HONEYMOONERS BEST CHOICE

HAVE A PLEASANT STAY IN ORANGE COUNTY

http://indiastudychannel.com/resources/44688-Orange-County.aspx

www.orangecounty.in/coorg/home.php

Vijay TV Neeya Naana

2009-11-28T07:46:00.000-08:00

This is regarding a programme called Neeya Naana? ( You or Me?), a debate show in Vijay TV. Those people who are interested to give your views and opinions and are really talented in arguing can participate in this show.

To take part in this show contact Kamaraj(9884845406), Executive producer of Neeya Naana.

Take part in this show and enjoy.

My baby Sachin Karthik Iyer

2009-11-28T06:37:00.000-08:00

Sachin is so cute. He was born on august 9, 2009. My chellam

கண் திறந்து பார்க்கும்

2009-10-27T01:54:00.000-07:00

கண் திறந்து பார்க்கும் அருள் தெய்வம் நீ அம்மா
இந்த மண்ணில் உன்னை மறவாத வாரம் வேண்டும் அம்மா
வர வேண்டும் கருணை நிதி வேண்டும்
நீ தர வேண்டும் நான் பெற வேண்டும்

விண்ணவர்க்கும் நீ விருந்தல்லவோ
மாரியம்மா உன் திருச்சாம்பல் மருந்தல்லவோ
கருணைமுகம் என்றும் காண வேண்டும்
உன் அருளாலே என் வாழ்வு மலர வேண்டும்

கருநாகமாய் நீ உரு மாறினாய்
திருவேற்காட்டில் ஒளிகொண்ட சுடராகினாய்
அருமறை பொருளான ஷிவஷக்தி கருமாரி
உன் அருளாலே என் வாழ்வு மலர வேண்டும்

Pachai mayil eri

2009-08-05T09:57:00.000-07:00

பச்சை மயில் ஏறி வாருமையா
பார்வதி பாலகனே
தேவாதி தேவனே
தேவர்க்கு அணுகுலனே (பச்சை)

அன்னை வள்ளி தெய்வானை யூடனே
அன்புடனே வந்த காந்தியனே
என்ன பாக்கியம் செய்தேனோ ஈசா உந்தனை காண
நேசா பழனிமலை வாசா கைலாசனே (பச்சை)

சூரனை சம்ஹாரம் செய்த
சுப்ரமணிய தண்டாயுதனே
தாரக சம்ஹரித்த சரவண ஷண்முகனே
சரணம் சரணம் உந்தன் கருணை கண்காட்சியே (பச்சை)

murugan songs tamil lyrics MURUGA UNAKKU PUGAZH MAALAI

2009-08-05T09:44:00.000-07:00

முருகன் பாடல்கள் 1
முருகா உனக்கு புகழ் மாலை
சூட்டுவதே என் முதல் வேலை
கந்தா உன் திரு வடிவேலை
வணங்கிட தானே அதிகாலை

தூவிட குறிஞ்சி மலருண்டு
தேன் தினையோடு கனியுண்டு
பாதத்தில் வைத்திட மனமுண்டு
பூஜையை ஏற்பாய் நீ வந்து

ஆலயம் என்பதுன் நிழல் தானே
அணையா தீபமுன் அருள் தானே
காலமும் துணையும் நீ தானே
கருணையை போழிவதுன் விழி தானே

தெய்வயானை ஒருபுறமும்
மான்மகள் வள்ளி மறுபுறமும்
தோன்றிட நீதரும் திருக்காட்சி
மங்களம் பெருகிடும் அருட்காட்சி

Microsoft Silverlight tutorials

2008-10-21T22:30:00.000-07:00

Microsoft Silverlight is one of the richest application experiences. It is an entrant to Adobe flash Player. It is based on the mark up language XAML. And it supports audio, video and animations. Beginners to Microsoft Silverlight can access http://www.dotnetspider.com/tutorials/Silverlight-Tutorials.aspx for tutorials. It consists of 28 chapters for easy learning.

Make money from Adsense

Lalitha Sahasranamam

2008-10-17T10:04:00.000-07:00

Sinduraruna vigraham trinayanam manikyamauli sphurat
Tara nayaka shekharam smitamukhi mapina vakshoruham
Panibhyamalipoorna ratna chashakam raktotpalam bibhratim
Saumyam ratna ghatastha raktacharanam dhyayet paramambikam --1

The Divine mother is to be meditated upon as shining in a vermilion-red body, with three eyes, sporting a crown of rubies studded with the crescent moon, a face all smiles, a splendid bust, one hand holding a jewel-cup brimming with mead, and the other twirling a red lotus.

Dhyayet padmasanastham vikasitavadanam padmapatrayatakshim
Hemabham pitavastram karakalitalasad hemapadmam varangim
Sarvalankara yuktam satata mabhayadam bhaktanamram bhavanim
Shrividyam shanta murttim sakala suranutam sarva sampatpradatrim ..3

The Divine Goddess is to be meditated upon as seated on the lotus with petal eyes. She is golden hued, and has lotus flowers in Her hand. She dispels fear of the devotees who bow before Her. She is the embodiment of peace, knowledge (vidyaa), is praised by gods and grants every kind of wealth wished for

Lalita Sahasranama stotram

Om shrimata shrimaharagyi shrimatsimha saneshvari
Chidagni kundasambhuta devakarya samudyata .. 1

Shrimata: Salutations to the Divine Mother, who is the Mother of all.
Shri-mahararagni: Great Empress of the whole Universe.
Shrimat-simhasaneshvari: Great Sovereign, enthroned on the lion’s back.
Chidagni kundasambhuta: Who came out of the fire of Pure Consciousness.
Devakarya samudyata: Who promotes the cause of Divine forces.

Udyadbhanu sahasrabha chaturbahu samanvita
Raagasvarupa pashadhya krodha karankushojjvala .. 2

Udyadbhanu sahasrabha: Who is radiant as a thousand suns rising together.
Chaturbahu samanvita: Four-armed Divinity.
Ragasvarupa pashadhya: Who holds in her lower left hand a noose representing the power of love.
Krodha karankushojjvala: Who holding the flashing Ankusa (goad) of anger in Her lower right hand for restraining the forces of evil.

Manorupekshu kodanda pancha tanmatra sayaka
Nijaruna prabhapura majjadbrahmanda mandala .. 3

Manorupekshu kodanda: Who wields in her upper left hand a Sugarcane bow that stands for mind.
Panchatanmatra sayaka: Who holds five arrows representing the five Tanmatras (Subtle elements).
Nijaruna prabhapura majjadbrahmanda mandala: In the rosy splendour of whose form the whole universe is bathed.

Champakashoka punnaga saugandhika lasatkacha
Kuruvinda manishreni kanatkotira mandita .. 4

Champakashoka punnaga saugandhika lasat kacha: Whose shining locks of hair impart their fragrance to flowers like Chamka, Ashoka and Punnaga adorning them.
Kurvinda manishreni kanatkotira mandita: Whose crown is shining with rows of Kuruvinda gems.

Ashtami chandra vibhraja dalikasthala shobhita
Mukhachandra kalankabha mruganabhi visheshaka .. 5

Ashtami chandra vibhraja dalikasthala shobhita: Whose forehead shines, arching like the crescent moon of the eighth lunar digit (Ashtami)
Mukhachandra kalankabha mruganabhi visheshaka: The Kasturi Tilaka adorns her moonlike face, like the spot in the moon.

Vadanasmara mangalya gruhatorana chillika
Vaktra lakshmi parivaha chalan minabha lochana .. 6

Vadanasmara mangalya gruhatorana chillika: Whose face, the auspicious home of Karma (Cupid), has eyebrows that resemble archways leading to that abode of beauty.
Vaktra lakshmi parivaha chalan minabha lochana: Whose eyes move like fish in the streams of beauty flowing from Her face.

Navachampaka pushpabha nasadanda virajita
Tarakanti tiraskari nasabharana bhasura .. 7

Navachampaka pushpabha nasadanda virajita: Whose shapely nose is like a freshly blown Champaka bud.
Tarakanti tiraskari nasabharana bhasura: With a nasal ornament set with a jewel that excels the brilliance of the planet Venus.

Kadamba manjari klupta karnapura manohara
Tatanka yugalibhuta tapanodupa mandala .. 8

Kadamba manjari klupta karnapura manohara: Who is radiant and charming with a bunch of Kadamba flowers over her ears.
Tatanka yugalibhuta tapanodupa mandala: Who has the orbs of the Sun and Moon as Her pair of ear pendants.

Padmaraga shiladarsha paribhavi kapolabhuh
Navavidruma bimbashri nyakkari radanachhada .. 9

Padmaraga shiladarsha paribhavi kapolabhuh: Whose cheeks are far fairer than mirrors of ruby (Padmaraga)
Navavidruma bimbashri nyakkari radanachhada: Whose lips outshine the redness of fresh coral and bimba fruit.

Shuddha vidyankurakara dvijapankti dvayojjvala
Karpura vitikamoda samakarshi digantara .. 10

Shuddha vidyankurakara dvijapankti dvayojjvala: Whose beauty is enhanced by her rows of teeth that resemble the sprouting of pure Knowledge (Suddha Vidya or Sri – Vidya).
Karpura vatikamoda samakarshi digantara: The fragrance of the campho limbedded betel roll in whose mouth is spreading in all directions.

Nijasallapa madhurya vinirbhartsita kachhapi
Mandasmita prabhapura majjatkamesha manasa .. 11

Nijasallapa madhurya vinirbhartsita kachhapi: Whose speech is more melodious than the Veena of Sarasvati known as Kachhapi.
Mandasmita prabhapura majjatkamesha manasa: The radiance of whose smile inundates the mind of Kamesvara, Her consort.

Anakalita sadrushya chubukashri virajita
Kamesha baddhamangalya sutra shobhita kandhara .. 12

Anakalita sadrushya chubukashri virajita: Her chin is peerless in beauty.
Kamesha baddhamangalya sutra shobhita kandhara: Whose neck is adorned with the Mangalasutra fastened thereon by Her consort Kamesvara.

Kanakangada keyura kamaniya bhujanvita
Ratnagraiveya chintaka lolamukta phalanvita .. 13

Kankangada keyura kamaniya bhujanvita: Whose beautiful arms are decked with armlets and bracelets of gold.
Ratnagraiveya chintaka lolamukta phalanvita: Who wears a gem – set necklace having a big pearl as a pendant.

Kameshvara premaratna manipratipana stani
Nabhyalavala romali lataphala kuchadvayi .. 14

Kamesvara premaratna manipratipana stani: Whose breasts form the price she pays to Her Consort (Mahesvara) in return for the gem of love He bestows on Her.
Nabhayalavala romali lataphala kuchadvayi: Whose breasts look like fruits on the creeper of the hair – line spreading upwards from the navel.

Lakshyaroma latadharata samunneya madhyama
Stanabhara dalanmadhya pattabandha valitraya .. 15

Lakshyaroma latadharata samunneya madhyama: Who has a waist so slender that it can only be inferred as a base for the creeper of fine hair springing from her navel upwards.
Stanabhara dalanmadhya pattabandha valitraya: Whose waist, breaking under the weight of the breasts, gets three lines like a supporting belt.

Arunaruna kausumbha vastrabhasvath katitati
Ratnakinkini karamya rashana dama bhushita .. 16

Arunaruna kausumbha vastrabhasvath katitati: Who wears a garment of deep red round Her hip.
Ratnakinkini karamya rashana dama bhushita: Who is adorned with a girdle having many mini bells set with precious stones.

Kamesha gyata saubhagya mardavoru dvayanvita
Manikya makutakara janudvaya virajita .. 17..

Kamesha gyata saubhagya mardavoru dvayanvita: The beauty and smoothness of whose thighs is known only to her consort, the Conqueror of love (Kamesha)
Manikya makutakara janudvaya virajita: Whose two knees are like crowns shaped from the precious stone Manikya.

Indragopa parikshipta smaratunabha janghika
Gudhagulpha kurmaprushta jayishnu prapadanvita .. 18

Indragopa parikshipta smaratunabha janghika: Whose calf – muscles resemble the quiver of the god of love with bright glow–worm like decorations.
Gudhagulpha: Whose ankled are well filled and therefore without protrusion.
Kurmaprushta jayishnu prapadanvita: The arch of whose feet rival the shapeliness and beauty of the back of a tortoise.

Nakhadidhiti sanchhanna namajjana tamogunaa
Padadvaya prabhajala parakruta saroruha .. 19

Nakhadidhiti sanchhanna namajjana tamoguna: The illumining splendor of whose tow nails dispel the darkness of ignorance in the votaries prostrating at Her feet.
Padadvaya prabhajala parakruta saroruha: Whose feet defeat the lotus in beauty.

Sinjana manimanjira manditashri padambuja
Marali mandagamana mahalavanya shevadhih .. 20

Sinjana manimanjira manditashri padambuja: Whose lotus – feet are adorned with tinkling anklets set with jewels.
Marali mandagamana: Whose gait is slow and gentle like that of a swan.
Mahalavanya shevadhih: Who is a treasure house of Divine beauty.

Sarvaruna navadyangi sarvabharana bhushita
Shiva kameshvarankastha shiva svadhina vallabha .. 21

Sarvaruna: Who is rose – hued all over.
Anavadyangi: Who is faultless in every limb.
Sarvabharana bhushita: Who is adorned with divine ornaments.
Shiva kameshvarankastha: Who is seated on the lap of Shiva, the conqueror of desire (Kamesvara).
Shiva: The Consort of Shiva, whose Power She is.
Svadhina vallabha: Who dominates over Her Consort Siva in the creative part of the cyclic motion of time.

Sumeru madhya shrungastha shrimannagara nayika
Chintamani gruhantastha panchabrahma sanasthita .. 22

Sumeru Madhya shrugastha: Who dwells on the mid most peak of Mount Meru.
Shrimannagara nayika: Who is the Bindu, the central circle of bliss in the Shri-chakra.
Chintamani gruhantastha: Whose abode is Manidvipa the Island of Wish-yielding Gem.
Panchabrahma sanasathita: Who rests on a seat formed of the five Divinities (Brahmans), Brahma, Vishnu, Rudra, Isana and Sadasiva.

Mahapadmatavi samstha kadamba vanavasini
Sudhasagara madhyastha kamakshi kamadayini .. 23

Mahapadmatavi samstha: Who dwells in a lotus forest, the thousand–petalled lotus of the Sahashra.
Kadamba vanavasini: Who resides amidst a grove of Kadamba trees (which fringe the Manidvipa)
Sudhasagara madhyastha: Who dwells in the center of the sea of Nectar (which is the light of the Blissful Moon of the pericap of the Sahashrara)
Kamakshi: The look of whose eyes is full of graceful
Kamadayini: Who grants all the prayers of votaries.

Devarshi ganasanghata stuyamanatma vaibhava
Bhandasura vadhodyukta shaktisena samanvita .. 24

Devarshi ganasanghata stuyamanatma vaibhava: Whose majesty is the subject of praise of hosts of sages and divine beings.
Bhadasura vadhodyukta shaktisena samanvita: Whose command an army of Saktis are intent on destroying Bhadasura (the Asura being Ignorance, Lalitambika, the Atman, and the Saktis, the potencies of the Atman.)

Sampatkari samarudha sindhura vrajasevita
Ashvarudha dhishtihitashva kotikoti bhiravruta .. 25

Sampatkari samarudha sindhura vrajasevita: Who is accompanied by a regiment of elephants, headed by Sampatkari.
Ashvarudha dhishtihitashva kotikoti bhiravruta: Who is surrounded by a cavalry of several crores of horses under the command of Ashvarudha.

Chakraraja ratharudha sarvayudha parishkruta
Geyachakra ratharudha mantrini parisevita .. 26

Chakraraja ratharudha sarvayudha parishkruta: Seated in Her chariot Chakra raja equipped with armaments of every kind.
Geyachakra ratharudha mantrini parisevita: Who is attended by Her minister (Syamala) seated in Her chariot named Geya – chakra.

Kirichakra ratharudha dandanatha purashkruta
Jvala malinikakshipta vahni prakara madhyaga .. 27

Kirichakra ratharudha dandanatha purashkruta: Who is preceded by Dandanatha, the commander of Her armiesin his chariot Kiri-chakra.
Jvala malinikakshipta vahni prakara madhyaga: Who has taken position in the center of the rampartof fire constructed by Jvalamalinika.

Bhandasainya vadhodyukta shaktivikrama harshita
Nitya parakramatopa nirikshana samutsuka .. 28

Bhadasainya vadhodyukta shaktivikrama harshita: Who rejoices at the valour of Her Saktis bent on destroying the army of Bhanda.
Nitya parakramatopa niriskhana samutsuka: Who is delighted on seeing the aggressiveness of Her Nitya deities in their attack on the army of Bhanda.

Bhandaputra vadhodyukta balavikrama nandita
Mantrinyamba virachita vishanga vadhatoshita .. 29

Bhandaputra vadhodyukta balavikrama nandita: Who is overjoyed to see Her daughter Bala who is intent on slaying the son of Bhanda.
Mantrinyamba virachita vishanga vadhatoshita: Who feels satisfaction at the destruction of Vishanga by Her chief minister Syamala.

Vishukra pranaharana varahi viryanandita
Kameshvara mukhaloka kalpita shriganeshvara .. 30

Vishukra pranaharana varahi viryanandita: Who appreciates the prowess displayed by Varahi in the destruction of Vishukra.
Kameshvara mukhaloka kalpita shriganeshvara: Who by a mere glance at her Consort Mahesvara generates Sri Ganesha (the elephant headed diety).

Mahaganesha nirbhinna vighnayantra praharshita
Bhandasurendra nirmukta shastra pratyastra varshini .. 31

Mahaganesha nirbhinna vighnayantra praharshita: Who rejoices when that Ganesha destroys the magical devices placed by Bhandasura as obstacles to Her victory.
Bhandasurendra nirmukta shastra pratyastra varshini: Who counters by Her own missiles the rain of missiles directed against Her by Bhadnasura.

Karanguli nakhotpanna narayana dashakrutih
Mahapashu patastragni nirdagdhasura sainika .. 32

Karanguli nakhotpanna rarayana dashakrutih: Who out of Her finger nails recreated all the ten Incarnations of Vishnu to destroy the Asuras slain by Him in His incarnations and now recreated magically by Bhanda out of a missile of his.
Mahapashu patastragni nirdaghasura sainika: Who burned to death the armies of demons with the fire of the great missile pashupata.

Kameshvarastra nirdagdha sabhandasura shunyaka
Brahmopendra mahendradi devasamstuta vaibhava .. 33

Kamesvarastra nirdagha sabhadasura shunyaka: Who with the flames of the missile Kamesvara caused the destruction of bandha and also of his Capita Sunyaka.
Brahmopendra mahendradi devasamstuta vaibhava: Whose manifold powers (displayed in the fight with Bhanda) are praised by Brahma, Vishnu and Indra.

Haranetragni sandagdha kama sanjiva naushadhih
Shrimadvagbhava kutaika svarupa mukhapankaja .. 34

Haranetragni sandagdha kama sanjiva naushadhih: The life giving herb, that revived the god of love (kama – deva) who had been burnt to death by the fire of Shiva’s eyes.
Shrimadvagbhava kutaika svarupa mukhapankaja: Whose lotus face represents the Vagbhava – Kuta of the pancha dashakshari – mantra which is subtle form of the Devi.

Kanthadhah katiparyanta madhyakuta svarupini
Shaktikutaika tapanna katyadho bhagadharini .. 35

Kanthadhah katiparyanta madhyakuta svarupini: Whose middle region from the neck to the waist is represented by the central part (Kamaraja – Kuta) of the same Mantra.
Shaktikutaika tapanna katyadho bhagadharini: Whose form below waist is identical with the last part (Sakti – Kuta) of Pancha – dashakshari Mantra.

Mulamantratmika mula kutatraya kalebara
Kulamrutaika rasika kulasanketa palini .. 36

Mulamantratmika: Who is the original Mantra (Mula-mantra, here Pancha – dashakshari) itself.
Mula-kuta-traya-kalebara: Whose body is identiacal with Pancha – Dashakshari Mantra with all its Kutas or combination of letters.
Kulamrutaika rasika: Who (as the Kundalini) revels in the nectar flowing from the Sahasrara through the whole of the Kula path (i.e.the Susumna).
Kulasanketa palini: Who guards the esoteric doctrine of the Kaulas.

Kulangana kulantastha kaulini kulayogini
Akula samayantastha samayachara tatpara .. 37

Kulangana: Who is the Female Element (Kundalini) in the Kula Path.
Kulantastha: Who is the innermost Reality of the Kula Path.
Kaulini: Who is called Kaulini, the core of the Kaula form of worship.
Kulayogini: Who is the Deity of the Kaulas.
Akula: Who is also the Akula (Siva) who is in the thousand-petalled lotus above the Kula Path.
Samayantastha: Who is likewise the center of the Samaya doctrine (in which the worship is done internally through meditation and which holds Siva-Sakti as of equal importance in all respects).
Samayanchara tatpara: Whom the Samaya tradition of worship is dear.

Muladharaika nilaya brahmagranthi vibhedini
Manipurantarudita vishnugranthi vibhedini .. 38

Muladharaika nilaya: Whose chief residence is the Muladhara.
Brahmagrandhi vibhedini: Who in Her ascent from the Muladhara breaks through the Brahma-grandhi (the Barrier of Brahma to the subtle dimension).
Manipurantarudita: Who then emerges in the Manipura – chakra.
Vishnugranthi vibhedini: Who then breaks through the Vishnu – granthi (the barrier to still subtler dimensions).

Agya chakrantaralastha rudragranthi vibhedini
Sahasraram bujarudha sudhasarabhi varshini .. 39

Agya chakrantaralstha: Who next abides in the center of the Agya – chakra.
Rudragrandthi vibhedini: Who finally breaks through the Rudra – granthi (the barrier to the subtlest dimension).
Sahasraram bujarudha: Who then ascends to the Thousand – petalled Lotus known as the Sahasrara.
Sudhasarabhi varshini: Who sends streams of Nectar (spiritual bliss) from the Transcendant moon in the Sahasrara.

Tadillata samaruchih shatchakropari samsthita
Mahasaktih kundalini bisatantu taniyasi .. 40

Tadillata samaruchih: Who shines like a steady flash of lightning.
Shatchakropari samsthita: Who then establishes herself above the six Chakras.
Mahasaktih: Whose immense joy consists in Asakti (union with Shiva)
Kundalini: Who resides in the Muladhara as the Kundalini (the coiled power).
Bisatantu taniyasi: Who is as fine and firm as the fibre of a lutus stalk.

Bhavani bhavanagamya bhavaranya kutharika
Bhadrapriya bhadramurtih bhakta saubhagya dayini .. 41

Bhavani: Who is Bhavani, the consort of Bhava (Shiva).
Bhavanagamya: Who is realized through devoted meditation.
Bhavaranya kutharika: Who is verily like an axe for clearing the jungle of Samsara (transmigratory existence) in which the Jiva is caught.
Bhadrapriya: Who is fond of everything auspicious.
Bhadramurtih: Who is the embodiment of auspiciousness.
Bhakta saubhagya dayini: Who grants all-round advancement to devotees, both in the spiritual and the material fields.

Bhaktipriya bhaktigamya bhaktivashya bhayapaha
Shambhavi sharadaradhya sharvani sharmadayini .. 42

Bhaktipriya: Who is fond of true devotion.
Bhaktigamya: Who is attained through true devotion.
Bhaktivashya: Who can be won over through true devotion.
Bhayapaha: Who dispels all fear.
Shambhavi: Who is known as Shambhavi, the Consort of Shiva.
Sharadaradhya: Who is adored by Sharada (the Consort of Brahma).
Sharvani: Who is the consort of Sharva or Shiva.
Sharmadayini: Who is the bestower of happiness.

Shankari shrikari sadhvi sharachandra nibhanana
Shatodari shantimati niradhara niranjana .. 43

Shankari: Who is Shankari the Consort of Shiva, who is inseparable from Her.
Shrikari: Who is the spouse of Vishnu, who brings prosperity to devotees.
Sadhvi: Who is a paragon of virtue.
Sharachandra nibhanana: Whose face shines like the autumnal moon.
Shatodari: Who has a very slender waist.
Shantimati: Who is full of peace.
Niradhara: Who has no support other than Herself. But supports everything else.
Niranjana: Who is free from the stain of ignorance.

Nirlepa nirmala nitya nirakara nirakula
Nirguna nishkala shanta nishkama nirupaplava .. 44

Nirlepa: Who is free from all affectations of external contacts.
Nirmala: Who is free from all impurities.
Nitya: Who is eternal.
Nirakara: Who is not limited to and by any form.
Nirakula: Who is never agitated.
Nirguna: Who is beyond the three Gunas of Prakrti – Sattva, Rajas and Tamas.
Nishkala: Who is the Partless Unitary Whole.
Shanta: Who is ever serene.
Nishkama: Who is free from desires.
Nirupaplava: Who is free from afflictions.

Nityamukta nirvikara nishprapancha nirashraya
Nityashuddha nityabuddha niravadya nirantara .. 45

Nityamukta: Who is eternally free.
Nirvikara: Who is not subject to change.
Nishprapancha: Who is beyond the sphere of multiplicity.
Nirashraya: Who is not dependent on anything or anyone.
Nityashuddha: Who is eternally pure.
Nityabuddha: Who is the ever – awake Consciousness.
Niravadya: Who is flawless.
Nirantara: Who is without any division, being eternal by nature.

Nishkarana nishkalanka nirupadhir nirishvara
Niraga ragamathani nirmada madanashini .. 46

Nishkarana: Who has no cause for Hersef, as she is the first cause.
Nishkalanka: Who is without any stain.
Nirupadhi: Who has no limitations.
Nirishvara: Who has no over – lord.
Niraga: Who is without passions.
Ragamathani: Who destroys all passion in the minds of devotees.
Nirmada: Who is without pride.
Madanashini: Who destroys all pride.

Nishchinta nirahankara nirmoha mohanashini
Nirmama mamatahantri nishpapa papanashini .. 47

Nishchinta: Who is free from all doubts and anxieties.
Nirahankara: Who is without any egoism.
Nirmoha: Who is free from false view of things.
Mohanashini: Who dispels all illusions.
Nirmama: Who is devoid of self-interest in any matter, as She includes everyting in Herself.
Mamatahantri: Who destroys the sense of self – centredness in devotees.
Nishpapa: Who is sinless.
Papanashini: Who destroys sins together with the root of all sinful tendencies.

Nishkrodha krodhashamani nirlobha lobhanashini
Nisamshaya samshayaghni nirbhava bhavanashini .. 48

Nishkrodha: Who is without anger.
Krodhashamani: Who destroys the tendency to get angry.
Nirlobha: Who is free from greed.
Lobhanashini: Who destroys greed in Her devotees.
Nisamshaya: Who has no doubts.
Samshayaghni: Who effaces all doubts.
Nirbhava: Who is not involved in the cycle of births and deaths.
Bhavanashini: Who frees devotees from involvement in the cycle of births and deaths.

Nirvikalpa nirabadha nirbheda bhedanashini
Nirnasha mrutyumathani nishkriya nishparigraha .. 49

Nirvikalpa: Who is without any modifications, as she is pure consciousness.
Nirabadha: Who can never be sublated.
Nirbheda: Who is beyond all differences.
Bhedanashini: Who destroys the sense of differences.
Nirnasha: Who is deathless.
Mrutymathani: Who destroys the fear of death.
Nishkriya: Who is without involvement in action.
Nishparigraha: Who needs no gift, as everything in the universe is hers.

Nistula nilachikura nirapaya niratyaya
Durlabha durgama durga duhkhahantri sukhaprada .. 50

Nistula: Who is peerless.
Nilachikura: Whose locks of hair are shining black colour.
Nirapaya: Who is imperishable.
Niratyaya: Who is indestructible.
Durlabha: Who is difficult to attain.
Durgama: Who is hard to approach.
Durga: Who is difficult of access.
Duhkhahantri: Who puts an end to sorrow.
Sukhaprada: Who bestows all happiness.

This artcle has been taken from http://www.astrojyoti.com/ls1.htm

2008-10-02T23:06:00.000-07:00

Sri Lakshmi Narasimha Karavalamba Stotram

2008-09-28T22:34:00.000-07:00

This sloka of shri Lakshmi Narasimha written by Adi Sankara gives protecting hands to those who are in need.

Sri Lakshmi Narasimha Karavalamba Stotram Lyrics - Lakshmi Nrsimha Prayer

Srimat payonidhi nikethana chakra pane,
Bhogeendra Bhoga mani rajitha punya moorthe,
Yogeesa saswatha saranya Bhabdhi potha,
Lakshmi Nrsimha Mama Dehi Karavalambam 1

Brahmendra, Rudra Arka kireeta koti,
Sangattithangri kamala mala kanthi kantha,
Lakshmi lasath kucha saroruha raja hamsa,
Lakshmi Nrsimha Mama Dehi Karavalambam 2

Samsara gora gahane charathe murare,
Marogra bheekara Mruga pravardhithasya,
Aarthasya mathsara nidha chain peedithasya,
Lakshmi Nrsimha Mama Dehi Karavalambam 3

Samsara koopam adhi ghora Magadha moolam,
Samprapya dukha satha sarpa samakulasya,
Dheenasya Deva krupana padamagadasya,
Lakshmi Nrsimha Mama Dehi Karavalambam 4

Samsara Sagara vishala karala kala,
Nakra graham grasana nigraha vigrahasya,
Vyagrasya raga rasanormini peedithasya,
Lakshmi Nrsimha Mama Dehi Karavalambam 5

Samasra Vrukshamagha bheeja manantha karma,
Sakha satham karana pathramananga pushpam,
Aroohasya dukha phalitham pathatho dayalo,
Lakshmi Nrsimha Mama Dehi Karavalambam 6

Samsara sarpa Ghana vakthra bhyogra theevra,
Damshtra karala visha daghdha vinashta murthe,
Naagari vahana sudhabhdhi nivasa soure,
Lakshmi Nrsimha Mama Dehi Karavalambam 7

Samsara dava dahanathura Bheekaroru,
Jwala valee birathi dhighdha nooruhasya,
Thwat pada padma sarasi saranagathasya,
Lakshmi Nrsimha Mama Dehi Karavalambam 8

Samsara Jala pathithasya Jagan nivasa,
Sarvendriyartha badisartha jashopamasya,
Proth Ganditha prachoora thaluka masthakasya,
Lakshmi Nrsimha Mama Dehi Karavalambam 9

Samsara bheekara kareeendra karabhigatha,
Nishpishta marmma vapusha sakalarthi nasa,
Prana prayana bhava bhhethi samakulasya,
Lakshmi Nrsimha Mama Dehi Karavalambam 10

Andhasya me viveka maha danasya,
Chorai prabho bhalibhi rindriya nama deyai,
Mohanda koopa kuhare vinipathathasya,
Lakshmi Nrsimha Mama Dehi Karavalambam 11

Baddhvaa gale yamabhataa bahutarjayantah,
Karshhanti yatra bhavapaashashatairyutam maam.
Ekaakinam paravasham chakitam dayaalo
Lakshmi Nrsimha Mama Dehi Karavalambam 12

Lakshmi pathe Kamala nabha suresa vishno,
Vaikunta Krishna Madhu soodhana pushkarksha,
Brahmanya kesava janardhana vasudeva,
Devesa dehi krupanasya karavalambam 13

Ekena chakramaparena karena shamkha-
Manyena sindhutanyaaamavalambya tishhthan,
Vaame karena varadaabhayapadmachihnam,
Lakshmi Nrsimha Mama Dehi Karavalambam 14

Samsaara saagara nimajjana muhyamaanam
diinam vilokaya vibho karunaanidhe maam,
prahlaada kheda parihaara paraavataara
Lakshmi Nrsimha Mama Dehi Karavalambam 15

Prahlaada naarada paraashara pundariika-
vyaasaadi bhaagavata pungavah rinnivaasa ,
Bhaktaanurakta paripaalana paarijaata,
Lakshmi Nrsimha Mama Dehi Karavalambam 16

Lakshhminrisimha charana abja madhuvratena
Stotram kritam shubhakaram bhuvi shankarena ,
Ye tatpathanti manujaa haribhakti yuktaa-
Ste yaanti tatpada saroja makhandaruupam 17

Sri Lakshmi Nrsimha Padarpanamasthu

For more slokas visit http://msabhiblog.blogspot.com/2008/08/sri-lakshmi-narasimha-karavalamba.html

http://www.geocities.com/ramya475/slns.htm

http://www.carnatica.net/lyrics/slokabank.htm

All Nokia Codes

2008-09-26T01:13:00.000-07:00

All Nokia Codes
NOKIA
Nokia Universal Codes
Code Description :
These Nokia codes will work on most Nokia Mobile Phones

1. *3370# Activate Enhanced Full Rate Codec (EFR) - Your phone uses the best sound quality but talk time is reduced my approx. 5%

2. #3370# Deactivate Enhanced Full Rate Codec (EFR) OR *3370#

3. *#4720# Activate Half Rate Codec - Your phone uses a lower quality sound but you should gain approx 30% more Talk Time.

4. *#4720# Deactivate Half Rate Codec.

5. *#0000# Displays your phones software version, 1st Line: Software Version, 2nd Line : Software Release

6. Date, 3rd Line: Compression Type.

7. *#9999# Phones software version if *#0000# does not work.

8. *#06# For checking the International Mobile Equipment Identity (IMEI Number).

9. (#pw+1234567890+ 1# Provider Lock Status. (Use the "*" button to obtain the "p,w" and "+" symbols).

10. #pw+1234567890+ 2# Network Lock Status. (Use the "*" button to obtain the "p,w" and "+" symbols).

11. #pw+1234567890+ 3# Country Lock Status. (use the "*" button to obtain the "p,w" and "+" symbols).

12. #pw+1234567890+ 4# SIM Card Lock Status. (use the "*" button to obtain the "p,w" Go to Top and "+" symbols).

13. *#147# (vodafone) this lets you know who called you last.

14. *#1471# Last call (Only vodofone).

15. *#21# Allows you to check the number that "All Calls" are diverted to

16. *#2640# Displays security code in use.

17. *#30# Lets you see the private number.

18. *#43# Allows you to check the "Call Waiting" status of your phone.

19. *#61# Allows you to check the number that "On No Reply" calls are diverted to.

20. *#62# Allows you to check the number that "Divert If Unreachable (no service)" calls are diverted to.

21. *#67# Allows you to check the number that "On Busy Calls" are diverted to.

22. *#67705646# Removes operator logo on 3310 & 3330.

23. *#73# Reset phone timers and game scores.

24. *#746025625# Displays the SIM Clock status, if your phone supports this power saving feature "SIM Clock Stop Allowed", it means you will get the best standby time possible.

25. *#7760# Manufactures code.

26. *#7780# Restore factory settings.

27. *#8110# Software version for the nokia 8110.

28. *#92702689# Displays - 1.Serial Number, 2.Date Made, 3.Purchase Date, 4.Date of last repair (0000 for no repairs), 5.Transfer User Data. To exit this mode you need to switch your phone off then on again. ( Favourite )

29. *#94870345123456789 # Deactivate the PWM-Mem.

30. **21*number# Turn on "All Calls" diverting to the phone number entered.

31. **61*number# Turn on "No Reply" diverting to the phone number entered.

32. **67*number# Turn on "On Busy" diverting to the phone number entered.

33. 12345 This is the default security code.

34. press and hold # Lets you switch between lines

35. NOKIA5110/5120/ 5130/5190

36. IMEI number: * # 0 6 #

37. Software version: * # 0 0 0 0 #

38. Simlock info: * # 9 2 7 0 2 6 8 9 #

39. Enhanced Full Rate: * 3 3 7 0 # [ # 3 3 7 0 # off]

40. Half Rate: * 4 7 2 0 #

41. Provider lock status: #pw+1234567890+ 1

42. Network lock status #pw+1234567890+ 2

43. Provider lock status: #pw+1234567890+ 3

44. SimCard lock status: #pw+1234567890+ 4

45. NOKIA 6110/6120/6130/ 6150/6190

46. IMEI number: * # 0 6 #

47. Software version: * # 0 0 0 0 #

48. Simlock info: * # 9 2 7 0 2 6 8 9 #

49. Enhanced Full Rate: * 3 3 7 0 # [ # 3 3 7 0 # off]

50. Half Rate: * 4 7 2 0 #

Check This Out & Exercise caution while answering mobile calls VERRRRRRRRY IMPPPPPORTTTANTT.

2008-09-26T00:30:00.000-07:00

This blog was an email received from my friend.

If you receive a phone call on your mobile from any person, saying that, he or she is a company engineer, or telling that they're checking your mobile line, and you have to press #90 or #09 or any other number.

End this call immediately without pressing any numbers. There is a fraud company using a device that once you press #90 or #09 they can access your "SIM" card and make calls at your expense.

Forward this message to as many friends as you can, to stop it.

All mobile users pay attention if you receive a phone call and your mobile phone displays (XALAN) on the screen don't answer the call, END THE CALL IMMEDIATELY, if you answer the call, your phone will be infected by a virus.

This virus will erase all IMEI and IMSI information from both your phone and your SIM card, which will make your phone unable to connect with the telephone network.

You will have to buy a new phone.

This information has been confirmed by both Motorola and Nokia.

There are over 3 Million mobile phones being infected by this virus in all around the world now.

You can also check this news in the CNN web site. The above news was also shown on the "INDIA TV" dated 07 th January 2008.

PLEASE FORWARD THIS PIECE OF INFORMATION TO ALL YOUR FRIENDS HAVING A MOBILE...

My Family

2008-09-24T01:24:00.000-07:00

SHIRDI SAI

2008-09-24T00:47:00.001-07:00

Sai Baba made eleven assurances to his devotees:

Whosoever puts their feet on Shirdi soil, their sufferings will come to an end.
The wretched and miserable will rise to joy and happiness as soon as they climb the steps of My Samadhi.
I shall be ever active and vigorous even after leaving this earthly body.
My tomb shall bless and speak to the needs of my devotees.
I shall be active and vigorous even from my tomb.
My mortal remains will speak from My tomb.
I am ever living to help and guide all who come to Me, who surrender to Me and who seek refuge in Me.
If you look to Me, I look to you.
If you cast your burden on Me, I shall surely bear it.
If you seek My advice and help, it shall be given to you at once.
There shall be no want in the house of My devotee.

For further information visit http://www.saibaba.org/

PCOS

2008-09-19T01:42:00.000-07:00

What is polycystic ovary syndrome (PCOS)?
Polycystic (pah-lee-SIS-tik) ovary syndrome (PCOS) is a health problem that can affect a woman's menstrual cycle, ability to have children, hormones, heart, blood vessels, and appearance. With PCOS, women typically have:

high levels of androgens (AN-druh-junz). These are sometimes called male hormones, although females also make them.
missed or irregular periods
many small cysts (sists) in their ovaries. Cysts are fluid-filled sacs.

How many women have polycystic ovary syndrome (PCOS)?
About one in ten women of childbearing age has PCOS. It can occur in girls as young as 11 years old. PCOS is the most common cause of female infertility (not being able to get pregnant).

What causes polycystic ovary syndrome (PCOS)?
The cause of PCOS is unknown. Most researchers think that more than one factor could play a role in developing PCOS. Genes are thought to be one factor. Women with PCOS tend to have a mother or sister with PCOS. Researchers also think insulin could be linked to PCOS. Insulin is a hormone that controls the change of sugar, starches, and other food into energy for the body to use or store. For many women with PCOS, their bodies have problems using insulin so that too much insulin is in the body. Excess insulin appears to increase production of androgen. This hormone is made in fat cells, the ovaries, and the adrenal gland. Levels of androgen that are higher than normal can lead to acne, excessive hair growth, weight gain, and problems with ovulation.

Does polycystic ovary syndrome (PCOS) run in families?
Most researchers think that PCOS runs in families. Women with PCOS tend to have a mother or sister with PCOS. Still, there is no proof that PCOS is inherited.

What are the symptoms of polycystic ovary syndrome (PCOS)?
Not all women with PCOS share the same symptoms. These are some of the symptoms of PCOS:

infrequent menstrual periods, no menstrual periods, and/or irregular bleeding
infertility (not able to get pregnant) because of not ovulating
increased hair growth on the face, chest, stomach, back, thumbs, or toes—a condition called hirsutism (HER-suh-tiz-um)
ovarian cysts
acne, oily skin, or dandruff
weight gain or obesity, usually carrying extra weight around the waist
insulin resistance or type 2 diabetes
high cholesterol
high blood pressure
male-pattern baldness or thinning hair
patches of thickened and dark brown or black skin on the neck, arms, breasts, or thighs
skin tags, or tiny excess flaps of skin in the armpits or neck area
pelvic pain
anxiety or depression due to appearance and/or infertility
sleep apnea—excessive snoring and times when breathing stops while asleep

Why do women with polycystic ovary syndrome (PCOS) have trouble with their menstrual cycle?
The ovaries are two small organs, one on each side of a woman's uterus. A woman's ovaries have follicles, which are tiny sacs filled with liquid that hold the eggs. These sacs also are called cysts. Each month about 20 eggs start to mature, but usually only one matures fully. As this one egg grows, the follicle accumulates fluid in it. When that egg matures, the follicle breaks open to release it. The egg then travels through the fallopian tube for fertilization. When the single egg leaves the follicle, ovulation takes place.

In women with PCOS, the ovary doesn't make all of the hormones it needs for any of the eggs to fully mature. Follicles may start to grow and build up fluid. But no one follicle becomes large enough. Instead, some follicles may remain as cysts. Since no follicle becomes large enough and no egg matures or is released, ovulation does not occur and the hormone progesterone is not made. Without progesterone, a woman's menstrual cycle is irregular or absent. Plus, the cysts make male hormones, which also prevent ovulation.

Does polycystic ovary syndrome (PCOS) change at menopause?
Yes and no. Because PCOS affects many systems in the body, many symptoms persist even though ovarian function and hormone levels change as a woman nears menopause. For instance, excessive hair growth continues, and male pattern baldness or thinning hair gets worse after menopause. Also, the risks of complications from PCOS, such as heart attack, stroke and diabetes, increase as a woman gets older.

What tests are used to diagnose polycystic ovary syndrome (PCOS)?
There is no single test to diagnose PCOS. Your doctor will take a medical history, perform a physical exam, and possibly take some tests to rule out other causes of your symptoms. During the physical exam the doctor will want to measure your blood pressure, body mass index (BMI), and waist size. He or she also will check out the areas of increased hair growth, so try to allow the natural hair growth for a few days before the visit. Your doctor might want to do a pelvic exam to see if your ovaries are enlarged or swollen by the increased number of small cysts. A vaginal ultrasound also might be used to examine the ovaries for cysts and check out the endometrium, the lining of the uterus. The uterine lining may become thicker if your periods are not regular. You also might have blood taken to check your hormone levels and to measure glucose (sugar) levels.

How is polycystic ovary syndrome (PCOS) treated?
Because there is no cure for PCOS, it needs to be managed to prevent problems. Treatment goals are based on your symptoms, whether or not you want to become pregnant, and lowering your chances of getting heart disease and diabetes. Many women will need a combination of treatments to meet these goals. Some treatments for PCOS include:

Birth control pills. For women who don't want to become pregnant, birth control pills can control menstrual cycles, reduce male hormone levels, and help to clear acne. However, the menstrual cycle will become abnormal again if the pill is stopped. Women may also think about taking a pill that only has progesterone, like Provera®, to control the menstrual cycle and reduce the risk of endometrial cancer. (See Does polycystic ovary syndrome (PCOS) put women at risk for other health problems?) But progesterone alone does not help reduce acne and hair growth.

Diabetes medications. The medicine metformin (Glucophage®) is used to treat type 2 diabetes. It also has been found to help with PCOS symptoms, although it is not FDA-approved for this use. Metformin affects the way insulin controls blood glucose (sugar) and lowers testosterone production. Abnormal hair growth will slow down, and ovulation may return after a few months of use. Recent research has shown metformin to have other positive effects, such as decreased body mass and improved cholesterol levels. Metformin will not cause a person to become diabetic.

Fertility medications. Lack of ovulation is usually the reason for fertility problems in women with PCOS. Several medications that stimulate ovulation can help women with PCOS become pregnant. Even so, other reasons for infertility in both the woman and man should be ruled out before fertility medications are used. Also, there is an increased risk for multiple births (twins, triplets) with fertility medications. For most patients, clomiphene citrate (Clomid®, Serophene®) is the first choice therapy to stimulate ovulation. If this fails, metformin taken with clomiphene is usually tried. When metformin is taken along with fertility medications, it may help women with PCOS ovulate on lower doses of medication. Gonadotropins (goe-NAD-oh-troe-pins) also can be used to stimulate ovulation. These are given as shots. But gonadotropins are more expensive and there are greater chances of multiple births compared to clomiphene. Another option is in vitro fertilization (IVF). IVF offers the best chance of becoming pregnant in any one cycle and gives doctors better control over the chance of multiple births. But, IVF is very costly.

Medicine for increased hair growth or extra male hormones. Medicines called anti-androgens may reduce hair growth and clear acne. Spironolactone (speer-on-oh-lak-tone) (Aldactone®), first used to treat high blood pressure, has been shown to reduce the impact of male hormones on hair growth in women. Finasteride (Propecia®), a medicine taken by men for hair loss, has the same effect. Anti-androgens often are combined with oral contraceptives.

Before taking Aldactone®, tell your doctor if you are pregnant or plan to become pregnant. Do not breastfeed while taking this medicine. Women who may become pregnant should not handle Propecia®.

Vaniqa® cream also reduces facial hair in some women. Other treatments such as laser hair removal or electrolysis work well at getting rid of hair in some women. A woman with PCOS can also take hormonal treatment to keep new hair from growing.

Surgery. "Ovarian drilling" is a surgery that brings on ovulation. It is sometimes used when a woman does not respond to fertility medicines. The doctor makes a very small cut above or below the navel and inserts a small tool that acts like a telescope into the abdomen. This is called laparoscopy. The doctor then punctures the ovary with a small needle carrying an electric current to destroy a small portion of the ovary. This procedure carries a risk of developing scar tissue on the ovary. This surgery can lower male hormone levels and help with ovulation. But these effects may only last a few months. This treatment doesn't help with loss of scalp hair and increased hair growth on other parts of the body.

Lifestyle modification. Keeping a healthy weight by eating healthy foods and exercising is another way women can help manage PCOS. Many women with PCOS are overweight or obese. Eat fewer processed foods and foods with added sugars and more whole-grain products, fruits, vegetables, and lean meats to help lower blood sugar (glucose) levels, improve the body's use of insulin, and normalize hormone levels in your body. Even a 10 percent loss in body weight can restore a normal period and make a woman's cycle more regular.

How does polycystic ovary syndrome (PCOS) affect a woman while pregnant?
There appears to be higher rates of miscarriage, gestational diabetes, pregnancy-induced high blood pressure (pre-eclampsia), and premature delivery in women with PCOS. Researchers are studying how the diabetes medicine metformin can prevent or reduce the chances of having these problems while pregnant. Metformin also lowers male hormone levels and limits weight gain in women who are obese when they get pregnant.

Metformin is a FDA pregnancy category B drug. It does not appear to cause major birth defects or other problems in pregnant women. But, there have been no studies of metformin on pregnant women to confirm its safety. Talk to your doctor about taking metformin during pregnancy or if you are trying to become pregnant. Also, metformin is passed through milk in breastfeeding mothers. Talk with your doctor about metformin use if you are a nursing mother.

Does polycystic ovary syndrome (PCOS) put women at risk for other health problems?

Women with PCOS have greater chances of developing several serious, life-threatening diseases, including type 2 diabetes, cardiovascular disease (CVD), and cancer. Recent studies found that:
More than 50 percent of women with PCOS will have diabetes or pre-diabetes (impaired glucose tolerance) before the age of 40.
Women with PCOS have a four to seven times higher risk of heart attack than women of the same age without PCOS.
Women with PCOS are at greater risk of having high blood pressure.
Women with PCOS have high levels of LDL (bad) cholesterol and low levels of HDL (good) cholesterol.

The chance of getting endometrial cancer is another concern for women with PCOS. Irregular menstrual periods and the absence of ovulation cause women to produce the hormone estrogen, but not the hormone progesterone. Progesterone causes the endometrium to shed its lining each month as a menstrual period. Without progesterone, the endometrium becomes thick, which can cause heavy bleeding or irregular bleeding. Over time, this can lead to endometrial hyperplasia, when the lining grows too much, and cancer.

I have PCOS. What can I do to prevent complications?
Getting your symptoms under control at an earlier age can help to reduce your chances of having complications like diabetes and heart disease. Talk to your doctor about treating all your symptoms, rather than focusing on just one aspect of your PCOS, such as problems getting pregnant. Also, talk to our doctor about getting tested for diabetes regularly. Eating right, exercising, and not smoking also will help to reduce your chances of having other health problems.

How can I cope with the emotional affects of PCOS?
Having PCOS can be difficult. Many women are embarrassed by their appearance. Others may worry about being able to get pregnant. Some women with PCOS might get depressed. Getting treatment for PCOS can help with these concerns and help boost a woman's self-esteem. Support groups located across the United States and on-line also can help women with PCOS deal with the emotional affects.;

For More Information...

You can find out more about PCOS by contacting the National Women's Health Information Center (NWHIC) at 1-800-994-9662 or the following organizations:

National Institute of Child Health and Human Development (NICHD), NIH, HHS
Phone: (800) 370-2943
Internet Address: http://www.nichd.nih.gov/womenshealth

American Association of Clinical Endocrinologists (AACE)
Phone: (904) 353-7878
Internet Address: http://www.aace.com

American Society for Reproductive Medicine (ASRM)
Phone: (205) 978-5000
Internet Address: http://www.asrm.org

Center for Applied Reproductive Science (CARS)
Phone: (423) 461-8880
Internet Address: http://www.ivf-et.com

InterNational Council on Infertility Information Dissemination, Inc. (INCIID)
Phone: (703) 379-9178
Internet Address: http://www.inciid.org

Polycystic Ovarian Syndrome Association, Inc. (PCOSA)
Internet Address: http://www.pcosupport.org

The Hormone Foundation
Phone: (800) 467-6663
Internet Address: http://www.hormone.org

This FAQ was reviewed by:

Louis V. DePaolo, Ph.D.
Chief, Reproductive Sciences Branch
Center for Population Research
National Institute of Child Health and Human Development

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2008-09-16T05:38:00.000-07:00

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Meaningful Roses

2008-09-15T01:35:00.000-07:00

Red Love, Beauty, Courage and Respect, Romantic Love, Congratulations, "I Love You", "Job Well Done", Sincere Love, Respect, Courage & Passion

Red (Dark) Unconscious beauty

Red (Single) "I Love You"

Pink Appreciation, "Thank you", Grace, Perfect Happiness, Admiration, Gentleness, "Please Believe Me"

Dark Pink Appreciation, Gratitude, "Thank You"

Light Pink Admiration, Sympathy, Gentleness, Grace, Gladness, Joy, Sweetness

White Purity, Innocence, Silence, Secrecy, Reverence, Humility, Youthfulness, "I am worthy of you", Heavenly

White (Bridal) Happy love

Orange Desire, Enthusiasm

Red and White Given together unity

Red and Yellow Jovial and Happy Feelings

Peach Appreciation, Closing the deal, Let's get together, Sincerity, Gratitude

Pale Peach Modesty

Coral Desire

Yellow Joy, Gladness, Friendship, Delight, Promise of a new beginning, Welcome Back, Remember Me, Jealousy, "I care"

Lavender Love at first sight, Enchantment

Orange Enthusiasm, Desire, Fascination

Single in any color Simplicity, Gratitude

Red Rosebud Symbolic of purity and loveliness

White Rosebud Symbolic of girlhood

Thornless Rose "Love at first sight"

Blue The unattainable, the impossible

Just Friendly

2008-09-12T07:51:00.000-07:00

Whole-cell simulation: a grand challenge of the 21st century Masaru Tomita

2008-09-11T22:11:00.000-07:00

Study of the cell will never be complete unless its dynamic behavior is understood. The complex behavior of the cell cannot be determined or predicted unless a computer model of the cell is constructed and computer simulation is undertaken. Rapid accumulation of biological data from genome, proteome, transcriptome and metabolome projects can bring us to the point where it is no longer purely speculative to discuss how to construct virtual cells in silico. This article describes attempts to construct whole cell models. The E-CELL project has completed a couple of virtual cell models, and computer simulations have revealed some biological surprises.

Suppose that a certain organism’s genome has been completely sequenced. Then uppose that structures and functions of all its gene products have been thoroughly identified. Suppose further that a giant map of the entire metabolic pathways has been drawn flawlessly. Then what? Would we have conquered the cell? The answer is clearly ‘no’ because the overall ‘behavior’ of the cell would still not be understood. To say that we understand the overall behavior of the cell, we must be able to answer questions such as: ‘How would the cell behave if we change the environment, for example, by adding or decreasing a certain substance?’ and ‘What is the result if a certain gene gets knocked out or over-expressed?’ Slightly more sophisticated questions include: ‘What gene needs to be inserted for the cell to behave in such a way’ and ‘What is the ideal culture medium in which to maximize the cell’s ability to do such a thing?’ There is no doubt that computer simulation is required to understand dynamic behaviors of cellular metabolism. Virtual experiments in computers, often called ‘in silico’ experiments are not just useful but indispensable for biology because of the large amount of data generated from the genome, proteome, transcriptome and metabolome projects.

‘Customized medicine’based on single nucleotide polymorphisms (SNPs) in which an appropriate dose of medicine is selected and administered to a specific patient based on his/her SNP data, is not an exception. When a large amount of SNP information from many different individuals is collected, the susceptibility of the patient to a certain drug can be statistically predicted by analysing a specific locus of a specific gene in his/her genome for SNPs. This statistical method is useful when a polymorphism of one specific gene causes a polymorphism of the phenotype (monogenic), in which case, analysis of the genotype can lead to a precise conclusion, without in silico experiments, as to what phenotype or physical trait that person has. It is not so easy, however, to understand a phenotype in which multiple genes are involved (polygenic). For example, suppose it has been shown statistically that a susceptibility to a certain drug is high (+) when a person possesses a type Agene; the susceptibility is also high (+) when possessing a type B gene at another locus. When a person has both Aand B gene types, however, there is no guarantee that his/her susceptibility to that drug will be twice as high (+/+); it is possible that his/her susceptibility would be low (−). Furthermore, suppose having a type C gene at a different locus is statistically known to result in low susceptibility (−). Then, what will happen when a patient has Aand C gene types? It is possible, for example, that the susceptibility is high (+) when the person only has A and C gene types but very low (−/−) if the person has a combination of B and C gene types, and high (+) again with combination of A, B and C, and so on. This non-monotonic nature of the genetic system is the reason why statistical analyses alone are limited in understanding cellular behaviors. Computer models and in silico experiments are necessary to understand and predict phenotypes of the cell, especially when they are polygenic phenotypes. After all, most biological and pathological phenomena in which the pharmaceutical industry has a great interest, such as cancer and allergy, are polygenic.

It is still an open question as to whether or not it is feasible to construct a computer model of a whole living cell that is sufficiently sophisticated to predict answers to the types of questions mentioned above. It is thought that the task of whole-cell modeling is too difficult to be achieved in the foreseeable future. Although no attempts were made towards whole-cell modeling until the late 1990s, the importance of computer simulation of cellular metabolisms has been suggested and emphasized since the 1980s. Cellular processes that are important subsystems of the cell have been modeled and simulated by many different groups: the regulation of gene expression1–5, cell cycle6,7, signal transduction8 and metabolic pathways9–12. However, although these models made significant contributions to the development of in silico biology, the programs were only able to handle specific subsystems, and it was difficult to combine different subsystem models into one single-cell model. The first cell model To conquer and directly challenge the task of whole-cell modeling, the E-CELL Project (Ref. 13) was initiated in 1996 at the Shonan-Fujisawa Campus of Keio University (Fujisawa, Japan), following the publication of the entire genome sequence of Mycoplasma genitalium http://www.tigr.org/tdb/mdb/mdbcomplete.html). M. genitalium has the smallest genome (580 kb) and the smallest number of genes (~480) of all living organisms currently known and its genomic sequences have been published (see http://www.tigr.org/). The size of its genome is one order of magnitude less than that
of Escherichia coli, and thus is an ideal candidate forwhole-cell modeling. Approximately 80% of its 480 genes share nucleotide sequence homology with other genes of which the function could be predicted when searched for on BLAST. The functions of the remaining 20% of the genes, however, are still unknown and it was soon realized that it would be difficult to reconstruct the whole organism without knowing the functions of
the remaining genes. However, it has been demonstrated in intensive gene-knock-out studies by The Institute for Genomic Research (TIGR) that many of the 480 genes are not always necessary for M. genitalium to survive. Therefore, in collaboration with TIGR, a minimal set of genes sufficient and necessary for its survival and for maintenance of its homeostasis were selected (Table 1) and the first hypothetical ‘virtual cell’with the 127 genes was constructed (Fig. 1). This virtual self-surviving cell (SSC) model takes up glucose into the cytoplasm, metabolizes the glucose through the glycolysis pathway and produces ATPas an energy source. The ATPis consumed mainly for protein synthesis; the 127 genes are transcribed by RNA polymerase into mRNAs, and then translated into proteins by ribosome. Proteins are modeled to degrade spontaneously over time and so the cell has to constantly produce protein to sustain life. The membrane structure of the cell is also modeled to degrade over time; thus, the cell has a phospholipid biosynthesis pathway for biosynthesis of the cell membrane, uptaking fatty acid and glycerol, consuming ATP and generating a phospholipid bilayer, which forms a cell membrane. A constant supply of energy (ATP) is required to maintain protein and membrane synthesis, and thus glucose is essential for the survival of the virtual cell.

E-CELL simulation system
The SSC model has 105 protein-coding genes (Table 2) and 22 RNA-coding genes, and consists of 495 reaction rules. Each reaction rule defines what to do within one single time step (one millisecond, in this case). Reactions include: (1) enzymatic reactions that increase and decrease the quantity of its substrate(s) and product(s), respectively; (2) complex formations, in which multiple substrates form a complex; (3) transportations that change the location of certain substances; and (4) stochastic processes such as a transcriptional factor binding to a specific site of the chromosome. The E-CELL system also accepts user-defined reactions, making it capable of handling many other phenomena such as diffusion and variable cell volume. When simulation of the SSC starts, all the reaction rules are executed in parallel (in practice pseudoparallel), and the overall behavior of the cell can be observed through various graphic interfaces (Fig. 2). The dynamic changes in the amount (molecular number) of various substances inside the cells can be observed using an interface (‘Tracer Window’, presented as ‘Traced substances’). In Fig. 2, C00031E and C00186E show temporal patterns of change in extracellular glucose and lactic acid (waste end product), respectively. It can be seen that the cell is ‘living’steadily because extracellular glucose is gradually decreasing and lactic acid is reciprocally increasing. If, at a certain time point, the extracellular glucose level is set to 0, the cell starts to starve and will eventually become incapable of glucose uptake. The activity of a specific biochemical reaction can be monitored using the ‘Reactor Window’(Fig. 2, ‘Reactor’ panel), which shows the number of molecules being processed within a single time step. The amount of substrates can be altered by the user, even in the middle of a simulation, using the ‘Substance Window’(Fig. 2, ‘Substance’panel). Finally, the expression of all genes can be monitored simultaneously using the ‘Genemap Window’(Fig. 2). Each icon represents each corresponding gene, and exhibits the amount of mRNA of that gene (molecular number). Aspecific gene can be easily knocked-out by clicking its icon; it is thus possible to conduct real-time knock-out experiments in silico. Clicking the icon again can reactivate a knocked-out gene. Final ’desperate efforts’ before starvation Even this simple cell model sometimes shows unpredictable behavior and has delivered biologically interesting surprises. When the extracellular glucose is drained and set to be zero, intracellular ATP momentarily increases and then decreases (Fig. 2, ‘Traced substances’ panel, substance ID C00002). At first, this finding was confusing. Because ATPis synthesized only by the glycolysis pathway, it was assumed that ATPwould decrease when the glucose, the only source of energy, becomes zero. After months of checking the simulation program and the cell model for errors, the conclusion is that this observation is correct and a rapid deprivation of glucose supplement can lead to the same phenomenon in living cells. The reason is, in fact, clear. In glycolysis, one molecule of glucose produces two molecules of ATP. In more detail, however, two molecules of ATP are consumed in the first part of glycolysis and then four molecules of ATP are synthesized in the second part, producing two net molecules of ATP. Thus, when glucose is rapidly deprived, first consumption of ATP stops and then, a few moments later, ATP synthesis stops. The slight difference in time causes a transient increase in ATP. This momentary increase in the level of ATP, is of great interest and is as if the cell undergoes a final ‘desperate effort’ when it is rapidly starved. Virtual erythrocytes obviously, the SSC model described above is only a hypothetical cell; no such cells exist in nature. Thus, it was decided to model living cells so that the simulation results could be evaluated. Human erythrocytes were chosen for the model because intracellular metabolism is limited in human erythrocytes and because they do not replicate, transcribe or translate genes; also, there are already several studies on the modeling of erythrocytes14–16. It is possible to compare computer models with real red blood cells because a considerable amount of experimental data about red blood cells has accumulated17, 18. Recently, the construction of a prototype of human erythrocytes using the E-CELL System has been completed (Fig. 3). In human erythrocytes, the major metabolic pathways are glycolysis, the pentose phosphate pathway and nucleotide metabolism. In addition, there are abundant hemoglobins that carry oxygen
from the lungs to peripheral tissues. Thus, the erythrocyte is a ‘bag’ packed with hemoglobins that maintain several metabolic pathways for ion transport to maintain homeostasis of intracellular osmosis. An E-CELL model of the human erythrocyte has been developed, by defining reaction rules for all these metabolisms based on the previous erythrocyte model14–17. All the kinetic equations and parameters used in the model were obtained from previously published experimental data. After tuning the kinetic parameters, the E-CELL erythrocyte model reached a steady state in which quantities of intermediate metabolites inside the virtual cell are comparable with the experimental data of living erythrocytes. We are currently extending and improving the erythrocyte model for a more accurate simulation, by taking into account osmotic pressure, pH and variable cell volume. Using the E-CELL for pathological analyses It is possible to perform in silico experiments in which
the function of an enzyme is inhibited, and to simulate the behavior of human erythrocytes from hereditary
anemic patients using the E-CELL model (Fig. 4). Using the simulated erythrocytes on the E-CELL program, the activity of aldolase is blocked in our virtual erythrocytes; aldolase (fructose bisphosphate aldolase) converts fructose-1,6-bis-phosphate (X12) to glyceraldehyde-3-phosphate (X14) and dihydroxyacetone- phosphate (X13). Aldolase-deficient humans are susceptible to hemolytic anemia. With aldolasedeficiency, the reactant fructose-1,6-bis-phosphate (X12) is markedly increased and accumulated, whereas the reaction products glyceraldehyde-3- phosphate (X14), dihydroxy-acetone-phosphate (X13) and further metabolites downstream of this reaction are significantly decreased. Although this is an expected result, such in silico experiments might give unexpected results, as we have seen in the SSC model. One major advantage of these in silico experiments is that they can be repeated automatically, for all enzymes at various levels of inhibition, resulting in lists of
enzyme deficiencies that could kill, perturb, or preserve the cell. The results might suggest some biologically
interesting phenomena, which could then be verified by in vitro or in vivo experiments. In addition, the results
would provide kinetic sensitivities of different enzymes. If a small change in enzyme activity results in a
dramatic change inside the cell, the enzymatic reaction is ‘sensitive’ and therefore would need accurate kinetic
parameter values. However, other enzymatic reactions that are not kinetically sensitive would need only rough values for their kinetic parameters. In this way, it will be possible to reproduce the behavior of human erythrocytes not only in normal physiological conditions but also in abnormal pathological conditions. Thus, one could investigate how an abnormal anemic condition occurs in human erythrocytes, for example, in patients with hereditary oxygen deficiency or patients with drug-induced inhibition of specific enzymes. Because there are many known cases of anemia with abnormal erythrocytes, computer simulation will have an important role in the development of remedies of many human diseases. SNPs and other types of genetic polymorphism
could result in different enzymes with different kinetic parameters, producing different fluxes and therefore
different metabolisms inside the cell. By assigning kinetic parameters based on SNP information, it might become possible to conduct ‘personalized simulation’ of an individual’s cellular metabolism. Such simulation could, in the future, play an important role in ‘customized medicine’, by predicting appropriate drugs and dosage for individual patients.

Future prospects
In addition to the ‘virtual self-surviving cell’ and the ‘human erythrocyte model’ described other E-CELL models are currently under construction; a ‘mitochondria model’ and a ‘signal transduction model’ for the chemotaxis of E. coli. Examples of other successful systems for integrative simulation of the cell include DBSolve by Goryanin and colleagues19 and the V-Cell by Schaff and co-workers20. One of the major problems in constructing large-scale cell models is lack of quantitative data. Most of the biological knowledge available is qualitative (such as functions of genes, pathway maps, which proteins interact with what) but for
simulation quantitative data (such as concentrations of metabolites and enzymes, flux rates, kinetic parameters and dissociation constants) are needed. Amajor challenge is to develop high-throughput technologies for measurement of inner-cellular metabolites. A large amount of data for a variety of cell states can then be collected with the technologies to construct quantitative models, and the models can be refined iteratively until the simulation results match the data. For this new type of simulation-orientated biology, we set up the Institute for Advanced Biosciences of Keio University (http://bioinfo.sfc.keio.ac.jp/IAB/). The institute consists of three centers for metabolome research, bioinformatics, and genome engineering, respectively. The ultimate goal of this international research institute is to construct a whole-cell model in silico based on a large amount of data generated by high-throughput metabolome analyses, and then to design a novel genome based on the computer
simulation and create real cells with the novel genome by means of genome engineering. In the USA, the National Institute of General Medicine Sciences (NIGMS) has announced the Alliance for Cellular Signaling (AFCS) for analyzing signal transduction of cardiomyocytes and B cells from the mouse. The ultimate goal of the project is ‘to create virtual cells’ of these particular cell types (http://www.nih.gov/news/pr/sep2000/nigms-05.htm). In addition, the Department of Energy (DOE) has set up a Microbial Cell Project (MCP), of which the
ultimate goal is to construct a ‘virtual microbe’ in silico (http://www.microbialcellproject.org), and the Special
Interest Group of Biological Simulation (SIGSIM) has been formed under the International Society for
Computational Biology (ISCB) for the purpose of effective communication among scientists working on
large scale cell modeling (http://www.iscb.org).

Summary
The cell is never ‘conquered’ until its total behavior is understood and the total behavior of the cell is never understood until it is modeled and simulated. Whole-cell modeling, which was thought intractable until recently, has suddenly become realistic. There is no doubt that in silico construction of complex living cells is an exciting scientific challenge and we are just opening the door to this new area of biological research in the 21st century.

Big Bang

2008-09-11T06:44:00.000-07:00

The Big Bang is the cosmological model of the universe that is best supported by all lines of scientific evidence and observation. The essential idea is that the universe has expanded from a primordial hot and dense initial condition at some finite time in the past and continues to expand to this day. Georges Lemaître proposed what became known as the Big Bang theory of the origin of the Universe, although he called it his 'hypothesis of the primeval atom'. The framework for the model relies on Albert Einstein's General Relativity as formulated by Alexander Friedmann. After Edwin Hubble discovered in 1929 that the distances to far away galaxies were generally proportional to their redshifts, this observation was taken to indicate that all very distant galaxies and clusters have an apparent velocity directly away from our vantage point. The farther away, the higher the apparent velocity.[1] If the distance between galaxy clusters is increasing today, everything must have been closer together in the past. This idea has been considered in detail back in time to extreme densities and temperatures, and large particle accelerators have been built to experiment on and test such conditions, resulting in significant confirmation of the theory. But these accelerators can only probe so far into such high energy regimes. Without any evidence associated with the earliest instant of the expansion, the Big Bang theory cannot and does not provide any explanation for such an initial condition, rather explaining the general evolution of the universe since that instant. The observed abundances of the light elements throughout the cosmos closely match the calculated predictions for the formation of these elements from nuclear processes in the rapidly expanding and cooling first minutes of the universe, as logically and quantitatively detailed according to Big Bang nucleosynthesis.

Fred Hoyle is credited with coining the phrase 'Big Bang' during a 1949 radio broadcast, as a derisive reference to a theory he did not subscribe to.[2] Hoyle later helped considerably in the effort to figure out the nuclear pathway for building certain heavier elements from lighter ones. After the discovery of the cosmic microwave background radiation in 1964, and especially when its collective frequencies sketched out a blackbody curve, most scientists were fairly convinced by the evidence that some Big Bang scenario must have occurred.